Results 171 to 180 of about 716,854 (348)

The Stellar Mass Function

open access: yes, 1997
15 pages incl. 4 figures (LaTeX, style files included); The figures are larger than in the 12 page review that is to appear in PASPC: Brown Dwarfs and Extrasolar Planets, R. Rebolo, M.R. Zapatero Osorio and E.
openaire   +2 more sources

The stellar initial mass function [PDF]

open access: yesProceedings of the International Astronomical Union, 2006
Abstract The IMF UNIVERSALITY HYPOTHESIS cannot be discarded despite the existence of the CLUSTER IMF THEOREM. This means that the currently existing star-formation theory fails to describe the stellar outcome. The IGIMF THEOREM, however, predicts a variation of galaxy-wide IMFs in dependence of the galaxy's star-formation rate even if the IMF ...
openaire   +1 more source

CCDC80 suppresses high‐grade serous ovarian cancer migration via negative regulation of B7‐H3

open access: yesMolecular Oncology, EarlyView.
PAX8 is a lineage‐specific master regulator of transcription in high‐grade serous ovarian cancer (HGSC) progression. We show for the first time that PAX8 facilitates proliferation and metastasis by repressing the cell autonomous tumor suppressor CCDC80 and inducing B7‐H3 expression.
Aya Saleh   +12 more
wiley   +1 more source

An Integrated Picture of Star Formation, Metallicity Evolution, and Galactic Stellar Mass Assembly

open access: yes, 2008
We present an integrated study of star formation and galactic stellar mass assembly from z=0.05-1.5 and galactic metallicity evolution from z=0.05-0.9 using a very large and highly spectroscopically complete sample selected by rest-frame NIR bolometric ...
A. J. Barger   +4 more
core   +1 more source

Cell‐cycle‐specific lesion evolution rather than inhibition of double‐strand‐break repair underpins cisplatin radiosensitization

open access: yesMolecular Oncology, EarlyView.
We analyze cisplatin–DNA adducts (CDAs) and double‐strand breaks (DSBs) in a cell‐cycle‐dependent manner. We find that CDAs form similarly across all cell cycle phases. DSBs arise only in S‐phase. CDAs might not directly impair DSB repair, but S‐phase DSB lesions evolve in the presence of CDAs and disrupt repair in G2, also causing radiosensitization ...
Ye Qiu   +10 more
wiley   +1 more source

Stellar-Mass Black Holes

open access: yesSymmetry
Stellar-mass black holes (3 M⊙≲MBH≲150 M⊙) are the natural product of the evolution of heavy stars (Mstar≳20 M⊙). In our Galaxy, we expect that 108–109 stellar-mass black holes have been formed from the gravitational collapse of heavy stars, but currently we know fewer than 100 objects.
openaire   +2 more sources

Revisiting Mission‐Oriented Cancer Research to tackle the increasing burden of cancer in Europe–a policy perspective

open access: yesMolecular Oncology, EarlyView.
Translational cancer research and its implementation through competitively selected Comprehensive Cancer Centers across Europe should be the primary policy focus for addressing the increasing cancer burden in Europe and counteract the present main strategy to convert cancer to a chronic disease.
Manuel Heitor   +2 more
wiley   +1 more source

The Lower Limit of Dynamical Black Hole Masses Detectable in Virgo Compact Stellar Systems Using the JWST/NIRSpec IFU

open access: yesThe Astrophysical Journal
Due to observational challenges, the mass function of black holes (BH) at lower masses is poorly constrained in the local universe. Understanding the occupation fraction of BHs in low-mass galaxies is crucial for constraining the origins of supermassive ...
Behzad Tahmasebzadeh   +5 more
doaj   +1 more source

The stellar populations of the bluest low surface brightness galaxies

open access: yes, 2005
Using optical/near-IR broadband photometry together with Halpha emission line data, we attempt to constrain the star formation histories, ages, total stellar masses and stellar mass-to-light ratios for a sample of extremely blue low surface brightness ...
Beijersbergen   +65 more
core   +1 more source

Metastasis on pause: How dormant tumor cells stay hidden within the tumor microenvironment and evade immune surveillance

open access: yesMolecular Oncology, EarlyView.
Dormant cancer cells can hide in distant organs for years, evading treatment and the immune system. This review highlights how signals from the surrounding tissue and immune environment keep these cells inactive or trigger their reawakening. Understanding these mechanisms may help develop therapies to eliminate or control dormant cells and prevent ...
Kanishka Tiwary   +1 more
wiley   +1 more source

Home - About - Disclaimer - Privacy