Results 211 to 220 of about 424,245 (266)

Molecular Mechanisms of Radioiodine Refractoriness in Differentiated Thyroid Cancer: Focus on Sodium/Iodide Symporter Dysregulation. [PDF]

Curr Issues Mol Biol
Rokashkov VD, Spirina LV, Tarasenko NV, Chizhevskaya SY.   +3 more
europepmc   +1 more source

Clinical and genotypic characteristics of 19 children with STXBP1-encephalopathy. [PDF]

Medicine (Baltimore)
Qi L, Fang C, Hu Z.
europepmc   +1 more source

Managing the rumen hydrogen economy to improve feed efficiency and climate-smart livestock production. [PDF]

Front Vet Sci
Kiadii O, Li L.
europepmc   +1 more source

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Genetics and Molecular Biology of Memory Suppression

The Neuroscientist, 2022
The brain is designed not only with molecules and cellular processes that help to form memories but also with molecules and cellular processes that suppress the formation and retention of memory. The latter processes are critical for an efficient memory management system, given the vast amount of information that each person experiences in their daily ...
Nathaniel C. Noyes, Ronald L. Davis
openaire   +2 more sources

The genetics of tumor suppression by p53

The FASEB Journal, 2022
Mutational inactivation of TP5 3 occurs commonly in cancer. In addition to mutation, there are over two hundred germline genetic variants in the TP5 3 coding region; some of these produce partially functional (hypomorphic) protein.
Maureen Murphy, Jessica Leung, Julia Leu, Alexandra Indeglia, Nicole Clarke, Toshitha Kannan, Nicole Kirven, Thibaut Barnoud, Andrew Kossenkov, Donna L. George   +9 more
openaire   +1 more source

Genetic Suppression of Polyglutamine Toxicity in Drosophila

Science, 2000
A Drosophila model for Huntington's and other polyglutamine diseases was used to screen for genetic factors modifying the degeneration caused by expression of polyglutamine in the eye. Among 7000 P-element insertions, several suppressor strains were isolated, two of which led to the discovery of the suppressor genes
Kazemi-Esfarjani, Parsa, Benzer, Seymour
openaire   +3 more sources

Nonsense suppression therapies in human genetic diseases

Cellular and Molecular Life Sciences, 2021
About 11% of all human disease-associated gene lesions are nonsense mutations, resulting in the introduction of an in-frame premature translation-termination codon (PTC) into the protein-coding gene sequence. When translated, PTC-containing mRNAs originate truncated and often dysfunctional proteins that might be non-functional or have gain-of-function ...
Patrícia Martins-Dias, Luísa Romão
openaire   +2 more sources