Results 121 to 130 of about 3,551 (191)

Characterization of Expression of the KCNE Gene Family in Zebrafish, Danio rerio [PDF]

open access: yes, 2011
The KCNE gene family codes for five transmembrane accessory proteins, minK related peptides or Mirps, involved in the modification of voltage-gated potassium (Kv) channels, K+ selective pores vital in the regulation of membrane potential and ...
Vernlund, Lauren
core   +1 more source

Characterizing the role of tafazzin in allergically activated mast cells [PDF]

open access: yes, 2020
Introduction & Aim: Allergic inflammatory diseases are a constantly growing health concern in westernized societies. Mast cells, the driving force behind many allergic diseases, modulate various metabolic pathways to carry out their various functions ...
Maguire, Aindriu
core   +1 more source

Corrigendum: Re-expression of tafazzin isoforms in TAZ-deficient C6 glioma cells restores cardiolipin composition but not proliferation rate and alterations in gene expression

open access: yesFrontiers in Genetics, 2022
Gayatri Jagirdar   +11 more
doaj   +1 more source

Stimulating the sir2–spargel axis rescues exercise capacity and mitochondrial respiration in a Drosophila model of Barth syndrome

open access: yesDisease Models & Mechanisms, 2022
Deena Damschroder   +5 more
doaj   +1 more source

Cross-species evidence for cardiolipin remodeling in neonatal hypoxic-ischemic encephalopathy. [PDF]

open access: yesJ Cereb Blood Flow Metab
Emaus KJ   +9 more
europepmc   +1 more source

Metabolic cardiomyopathies: untangling clinical heterogeneity with human stem-cell derived models. [PDF]

open access: yesEMBO Mol Med
Passadouro AS   +6 more
europepmc   +1 more source

Granulopoietic Dysregulation in a Patient-Tailored Mouse Model of Barth Syndrome. [PDF]

open access: yesStem Cell Rev Rep
Sierra Potchanant EA   +12 more
europepmc   +1 more source

Disturbed mitochondrial maturation in cardiolipin remodeling-deficient cardiomyocytes. [PDF]

open access: yesiScience
Senoo N   +6 more
europepmc   +1 more source

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