Results 181 to 190 of about 189,138 (299)
Whisker stimulation reinforces a resting-state network in the barrel cortex: Nested oscillations and avalanches. [PDF]
Mariani B +6 more
europepmc +1 more source
Abstract Objective Although the centromedian nucleus of the thalamus (CM) is an increasingly considered deep brain stimulation (DBS) target for drug‐resistant epilepsy (DRE), there is significant variability in programming practices, which may contribute to heterogenous outcomes.
Mohammed A. AlQahtani +7 more
wiley +1 more source
FcγR- and CD9-dependent synapse-engulfing microglia in the thalamus drive cognitive impairment following cortical brain damage in mice. [PDF]
Matoba K +15 more
europepmc +1 more source
Thalamus caudal, habenula. VPM, VPL, centromedian nucleus, red nucleus. Transverse plane.
core
Abstract Objective Resective surgery achieves seizure freedom in approximately 60%–80% of patients with drug‐resistant temporal lobe epilepsy (TLE), yet the extent to which seizure cessation permits recovery of disrupted functional brain networks remains unclear.
Addison C. Cavender +15 more
wiley +1 more source
Frequency-dependent effects of motor thalamus deep brain stimulation on speech and swallowing. [PDF]
Tang LW +21 more
europepmc +1 more source
Abstract Objective In temporal lobe epilepsy (TLE), patients often present with neurobehavioral comorbidities encompassing affective and cognitive difficulties. Whereas the latter have been related to atypical connectivity of mesiotemporal and frontotemporal circuits, the brain basis of affective symptoms remains incompletely understood.
Fatemeh Fadaie +4 more
wiley +1 more source
Hierarchically and Temporally Distinct Ca<sup>2+</sup> Signaling Across Thalamic Nuclei During Nociceptive Stimulation. [PDF]
Liu YH +10 more
europepmc +1 more source
Inherited metabolic epilepsies–established diseases, new approaches
Abstract Inherited metabolic epilepsies (IMEs) represent the inherited metabolic disorders (IMDs) in which epilepsy is a prevailing component, often determining other neurodevelopmental outcomes associated with the disorder. The different metabolic pathways affected by individual IMEs are the basis of their rarity and heterogeneity.
Itay Tokatly Latzer, Phillip L. Pearl
wiley +1 more source

