Results 161 to 170 of about 523,717 (316)

The mutation Ala677->Val in the methylene tetrahydrofolate reductase gene: A risk factor for arterial disease and venous thrombosis

open access: yes, 2015
Mild hyperhomocysteinemia has been identified as a risk factor for arterial disease and for venous thrombosis. Individuals homozygous for the thermolabile variant of the methylene tetrahydrofolate reductase gene (MTHFR) which results from a common ...
AnnichinoBizzacchi, JM   +4 more
core  

Striking Bone Marrow Plasmacytosis Resulting From Visceral Leishmaniasis

open access: yes
American Journal of Hematology, EarlyView.
Vishakha Sovani   +2 more
wiley   +1 more source

Clinical and Laboratory Characterization of Acquired Von Willebrand Syndrome

open access: yesAmerican Journal of Hematology, EarlyView.
ABSTRACT Acquired von Willebrand Syndrome (AVWS) is a rare bleeding disorder characterized by quantitative or qualitative defects of von Willebrand factor (VWF) in patients without a personal or family history of bleeding. It is frequently associated with systemic diseases, particularly lymphoproliferative disorders (LPDs) and myeloproliferative ...
Alessandro Ciavarella   +10 more
wiley   +1 more source

185 The Youth is our Future, and the Future is Here

open access: yesResearch and Practice in Thrombosis and Haemostasis
Omkar Garud   +4 more
doaj   +1 more source

Treatment of Acute M3 Thrombosis. [PDF]

open access: yesJACC Case Rep
Syed BH   +11 more
europepmc   +1 more source

Therapeutic Outcomes in VEXAS Syndrome: A Multicenter Comparative Cohort of Allogeneic Hematopoietic Stem Cell Transplantation and Hypomethylating Agents

open access: yesAmerican Journal of Hematology, EarlyView.
Key findings of therapeutic outcomes from a multi‐center cohort of VEXAS patients. ABSTRACT Hypomethylating agents (HMA) and allogeneic hematopoietic stem cell transplantation (alloHSCT) have both demonstrated remissions in VEXAS; however, comparative data is lacking.
Saubia Fathima   +48 more
wiley   +1 more source

180 Elevation of D-Dimer Without Any Evidence of Venous Thromboembolism: What Is Your Interpretation?

open access: yesResearch and Practice in Thrombosis and Haemostasis
Om Joshi   +4 more
doaj   +1 more source

Molecular mechanism of ischemic postconditioning in promoting diabetic ischemic brain injury repair via the microRNA‐34a–BDNF–SIX3 signaling axis

open access: yesAnimal Models and Experimental Medicine, EarlyView.
Diabetes combined with ischemic stroke (DMIS) exacerbates brain infarct size and neuronal damage compared to nondiabetic ischemic stroke (IS). This study reveals that microRNA‐34a (miR‐34a) plays a key role in DMIS pathogenesis: miR‐34a directly targets and suppresses brain‐derived neurotrophic factor (BDNF) and Sine oculis homeobox 3 (SIX3), promoting
Ling Zhao   +5 more
wiley   +1 more source

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