Results 171 to 180 of about 395,580 (215)
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Thrombocytosis and Thrombosis

Hematology, 2007
Abstract The aim of this review is to discuss current diagnostic approaches to, and classification of, patients presenting with thrombocytosis, in light of novel information derived from the discovery of specific molecular abnormalities in chronic myeloproliferative disorders (CMPD), which represent the most common cause of primary ...
VANNUCCHI, ALESSANDRO MARIA, Barbui T.
openaire   +4 more sources

Perianal thrombosis

Diseases of the Colon & Rectum, 1988
Two patients who had suffered numerous previous attacks of perianal hematoma were treated by excision of the lesions and adjacent skin (low hemorrhoidectomy). Histology confirmed that these lesions are not hematomas but thrombi lying within the thin-walled vessels of the external anal plexus.
S, Brearley, R, Brearley
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THROMBOSIS AND ATHEROSCLEROSIS

British Medical Bulletin, 1978
From the clinical point of view the most important end results of atherosclerosis are stenosis and occlusion of the affected artery. A prerequisite for this is a degree of plaque growth sufficient to encroach significantly on the integrity of the vessel lumen.
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Hypercoagulability And Thrombosis

Medical Clinics of North America, 1994
This article has stressed the common hereditary and acquired blood protein defects associated with thrombosis. The commonest hereditary defects appear to be antithrombin, protein C, and protein S deficiency, and the commonest acquired defects are anticardiolipin antibodies and the lupus anticoagulant.
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The Endothelium and Thrombosis

Seminars in Thrombosis and Hemostasis, 2000
Thrombosis reflects an imbalance between procoagulant and anticoagulant mechanisms. In some cases, thrombotic lesions are precipitated by gross changes in blood flow, vascular wall integrity, or systemic levels of coagulation factors. In other cases, thrombosis is induced by functional changes within the endothelium.
P L, Gross, W C, Aird
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Aging and Thrombosis

Seminars in Thrombosis and Hemostasis, 2002
Advanced age is associated with a dramatic increase in the rates of venous and arterial thrombotic events. Increases in fibrinogen, factors VIII and IX, and other coagulation proteins, without a proportional increase in anticoagulant factors, likely contribute to this risk.
Wade R, Wilkerson, David C, Sane
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Catheter Thrombosis

Seminars in Dialysis, 2001
Catheter malfunction due to poor flow is a common problem. When it occurs early, the cause is generally technical. Late occurrences are most often related to thrombus formation. Several types of thrombus may be seen, differing by location and supposed mechanism of formation. The most common offender, however, is the fibrin sheath thrombus.
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Childhood Thrombosis

Pediatrics, 1995
Objective. The objective of our study was to evaluate the age, sex, clinical conditions, family history, site, catheter association, means of radiologic evaluation, development of pulmonary involvement, prevalence of antithrombin III, protein C and protein S deficiencies, and lupus anticoagulants in children who suffered a thrombotic event.
R, Nuss, T, Hays, M, Manco-Johnson
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Cytokines and Thrombosis

Journal of Cardiovascular Pharmacology, 1995
Cytokines are pleiotropic mediators of inflammation and immunity. Leukocytes and vascular cells are both sources of cytokines and targets for them. Several cytokines affect key functions of vascular wall cells. Several clinical acute or chronic inflammatory situations associate modifications of the cytokine network and a prothrombotic state.
C, Dosquet, D, Weill, J L, Wautier
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Leukocytes and Thrombosis

Thrombosis and Haemostasis, 1965
SummaryWhite blood cells can no longer be considered simple trapped inclusions within thrombi. Their numbers in thrombi relative to blood counts increase with time. They appear to come from the blood flowing past the thrombus. They appear to migrate by amoeboid movement into the thrombic mass.
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