Results 71 to 80 of about 5,025 (225)

Silent Presentation, Significant Threat: Cold Agglutinin Syndrome After Haploidentical Hematopoietic Stem Cell Transplantation

open access: yeseJHaem, Volume 7, Issue 2, April 2026.
This graphic abstract presents a comprehensive overview of cold agglutinin syndrome (CAS) following haploidentical hematopoietic stem cell transplantation (haplo‐HSCT) based on the content of our letter, including its clinical scenarios, potential triggers, pathophysiological mechanisms, clinical manifestations, as well as therapeutic and management ...
Zhuoyu An   +18 more
wiley   +1 more source

PROTEOLYTIC PROCESSING OF VON WILLEBRAND FACTOR BY ADAMTS13 AND LEUKOCYTE PROTEASES-rev1-MJHID-2013

open access: yesMediterranean Journal of Hematology and Infectious Diseases, 2014
ADAMTS13 is a 190 kDa zinc protease encoded by a gene located on chromosome 9q34.   This protease specifically hydrolyzes von Willebrand factor (VWF) multimers, thus causing VWF size reduction.
Raimondo De Cristofaro
doaj  

Fostamatinib treatment for patients with antiphospholipid syndrome and low platelet count: A case series

open access: yes
British Journal of Haematology, EarlyView.
Ekaterina Balaian   +6 more
wiley   +1 more source

Rapidly Progressive Kidney Failure With Transient Non‐Cystic Kidney Enlargement: A Case Report Highlighting Delayed Medullary Cyst Formation

open access: yesNephrology, Volume 31, Issue 4, April 2026.
ABSTRACT Hereditary tubulointerstitial kidney diseases typically manifest as slowly progressive chronic kidney disease. Rapidly progressive kidney failure with non‐cystic nephromegaly is an exceptionally rare presentation posing significant diagnostic challenges.
Hiroki Ito   +11 more
wiley   +1 more source

Cancer-related thrombotic microangiopathies: case report and narrative review

open access: yesItalian Journal of Medicine, 2018
Thrombotic manifestations are common in cancer patients and increase morbidity and mortality. They can affect large or small, venous or arterial vessels. Thrombosis of large arteries and veins is much more studied than that of small vessels. Nevertheless,
Micaela La Regina   +5 more
doaj   +1 more source

Automating Vascular Biology: An End‐to‐End Automated Workflow for High‐Throughput Blood Vessel‐on‐a‐Chip Production and Multi‐Site Validation

open access: yesAdvanced Healthcare Materials, Volume 15, Issue 12, 25 March 2026.
AngioPlate384 is a 384‐well open‐top platform that automates production of more than 100 miniaturized, perfusable blood vessels embedded in hydrogel and supported by stromal cells. Stromal‐endothelial co‐culture strengthens blood vessel barrier function and yields responses useful for translational planning. Scalable and automation‐ready, it suits drug
Dawn S. Y. Lin   +14 more
wiley   +1 more source

A Novel Modified Bu/Vp16/cy/Flu/Ara‐C Conditioning Regimen Enhances Outcomes for High‐Risk Acute Lymphoblastic Leukemia Patients Undergoing Allogeneic Hematopoietic Stem Cell Transplantation

open access: yesCancer Medicine, Volume 15, Issue 3, March 2026.
ABSTRACT Purpose Allogeneic hematopoietic stem cell transplantation (allo‐HSCT) effectively treats high‐risk acute lymphoblastic leukemia (ALL), yet challenges persist due to post‐transplant relapse and conditioning regimen toxicities. The determination of an appropriate preconditioning regimen is critical to improving patient outcomes.
Xiaoyan Zhao   +6 more
wiley   +1 more source

Cardiac involvement in thrombotic microangiopathies

open access: yesТерапевтический архив, 2015
Aim. To describe cardiac involvement in patients with acute thrombotic microangiopathy (TMA). Materials and methods. The case histories of 46 patients with proven TMA, including 17 patients diagnosed with atypical hemolytic uremic syndrome (aHUS) and 29 ...
D S Tuter   +6 more
doaj  

Hyperfunctional complement C3 promotes C5-dependent atypical hemolytic uremic syndrome in mice [PDF]

open access: yes, 2019
Atypical hemolytic uremic syndrome (aHUS) is frequently associated in humans with loss-of-function mutations in complement-regulating proteins or gain-of-function mutations in complement-activating proteins.
Atkinson, John P   +11 more
core   +3 more sources

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