Results 31 to 40 of about 38,461 (234)

Thyrotropin-releasing hormone (TRH) promotes wound re-epithelialisation in frog and human skin

open access: yes, 2013
There remains a critical need for new therapeutics that promote wound healing in patients suffering from chronic skin wounds. This is, in part, due to a shortage of simple, physiologically and clinically relevant test systems for investigating candidate ...
Paredes Roberto   +59 more
core   +1 more source

Carbamazepine and Thyroid Hormones

open access: yesPediatric Neurology Briefs, 2001
Changes in serum thyroid hormone levels in 37 children with epilepsy receiving carbamazepine (CBZ) and valproic acid (VPA) were analyzed at the University of Chieti and University of Siena, Italy.
J Gordon Millichap
doaj   +1 more source

Electrophysiological Effects of Ghrelin in the Hypothalamic Paraventricular Nucleus Neurons

open access: yesFrontiers in Cellular Neuroscience, 2018
The paraventricular nucleus (PVN) is involved in the control of sympathetic tone and the secretion of hormones, both functions known to be influenced by ghrelin, suggesting direct effect of ghrelin in this nucleus.
Raoni C. dos-Santos   +5 more
doaj   +1 more source

Effect of medroxyprogesterone acetate on thyrotropin secretion in adult and old female rats

open access: yesBrazilian Journal of Medical and Biological Research, 2000
Steroid hormones have been implicated in the modulation of TSH secretion; however, there are few and controversial data regarding the effect of progesterone (Pg) on TSH secretion. Medroxyprogesterone acetate (MPA) is a synthetic alpha-hydroxyprogesterone
R.M. Moreira   +5 more
doaj   +1 more source

Thyrotropin-Releasing Hormone (TRH) : An Alternative Therapy for Infantile Spasms

open access: yesPediatric Neurology Briefs, 1987
Pediatric neurologists at the Central Hospital, Aichi Prefectural Colony, Kasugai, Aichi 480-03, Japan, compared the effects of TRH in 31 children and ACTH in 33 with severe epilepsy.
J Gordon Millichap
doaj   +1 more source

Linkage and association of rs3110045 and rs28499085 variants in the thyrotropin-releasing hormone receptor (TRHR) gene with the risk of familial type 2 diabetes

open access: yesAspects of Molecular Medicine
Type 2 diabetes (T2D) is a chronic and prevalent multisystemic disease that significantly increases morbidity and mortality. Dysfunction of the thyroid hormone system is common in patients with T2D, increasing their risk of both hyperthyroidism and ...
Rongling Wu, Claudia Gragnoli
doaj   +1 more source

PVN pathways controlling energy homeostasis

open access: yesIndian Journal of Endocrinology and Metabolism, 2012
Research into the control of energy balance has tended to focus on discrete brain regions, such as the brainstem, medulla, arcuate nucleus of the hypothalamus, and neocortex.
Jennifer W Hill
doaj   +1 more source

Neonatal screening and a new cause of congenital central hypothyroidism [PDF]

open access: yesAnnals of Pediatric Endocrinology & Metabolism, 2014
Congenital central hypothyroidism (C-CH) is a rare disease in which thyroid hormone deficiency is caused by insufficient thyrotropin (TSH) stimulation of a normally-located thyroid gland.
Toshihiro Tajima   +3 more
doaj   +1 more source

Involvement of the hypothalamic-pituitary-thyroid axis and its interaction with the hypothalamic-pituitary-adrenal axis in the ontogeny of avian thermoregulation: a review

open access: yes, 2008
The emergence of thermoregulation in avian species is a complex matter in which neural as well as hormonal processes are involved. In a previous paper, the neural aspects of primary avian thermoregulation were discussed.
Brand, H., van den   +5 more
core   +1 more source

Natural Resistance to Ovarian Hyperstimulation Syndrome in Estrildid Finches Reveals Macrophage GPR183 as a Potential Therapeutic Target

open access: yesAdvanced Science, EarlyView.
Ovarian macrophage depletion reverses OHSS resistance in estrildid finches and exacerbates OHSS symptoms in rats. Activating macrophage GPR183 alleviates OHSS by reducing pro‐inflammatory factors, increasing immunomodulatory molecules, remodeling CD44/SDC4‐mediated communication, and restoring immune homeostasis.
Xiaofei Yan   +11 more
wiley   +1 more source

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