Results 11 to 20 of about 66,421 (262)

Secondary amenorrhea in a woman with spinocerebellar degeneration treated with thyrotropin-releasing hormone: a case report and in vitro analysis

open access: yesJournal of Medical Case Reports, 2011
Introduction While thyrotropin-releasing hormone is known to be a prolactin-release stimulating factor, thyrotropin-releasing hormone-tartrate and its derivative, taltirelin hydrate, are used for the treatment of spinocerebellar degeneration, a ...
Kanasaki Haruhiko   +4 more
doaj   +1 more source

Thyroid hormone therapy for older adults with subclinical hypothyroidism [PDF]

open access: yes, 2017
The Authors ...
Bauer, Douglas C.   +2 more
core   +2 more sources

A Causality between Thyroid Function and Bone Mineral Density in Childhood: Abnormal Thyrotropin May Be Another Pediatric Predictor of Bone Fragility

open access: yesMetabolites, 2023
Low bone mass can occur in children and adolescents with numerous chronic conditions; however, the influence of abnormal thyroid hormone and thyroid-stimulating hormone (TSH) levels on low bone mineral density (BMD) in children and adolescents remains ...
Dongjin Lee, Moon Bae Ahn
doaj   +1 more source

TRH: Pathophysiologic and clinical implications [PDF]

open access: yes, 1985
Thyrotropin releasing hormone is thought to be a tonic stimulator of the pituitary TSH secretion regulating the setpoint of the thyrotrophs to the suppressive effect of thyroid hormones. The peptide stimulates the release of normal and elevated prolactin.
A. Enjalbert   +82 more
core   +1 more source

The Usefulness of Thyroid Antibodies in the Diagnostic Approach to Autoimmune Thyroid Disease

open access: yesAntibodies, 2023
Autoimmune thyroid disease (AITD) refers to a spectrum of various diseases, with two extremes of clinical presentation, hypothyroidism (Hashimoto’s thyroiditis (HT) and hyperthyroidism (Graves–Basedow disease (GBD)).
Hernando Vargas-Uricoechea   +3 more
doaj   +1 more source

The Pathogenic TSH β-Subunit Variant C105Vfs114X Causes a Modified Signaling Profile at TSHR [PDF]

open access: yes, 2019
1) Background: Central congenital hypothyroidism (CCH) is a rare endocrine disorder that can be caused by mutations in the β-subunit of thyrotropin (TSHB).
Biebermann, Heike   +6 more
core   +1 more source

A Genomic Point Mutation in the Extracellular Domain of the Thyrotropin Receptor in Patients with Graves’ Ophthalmopathy [PDF]

open access: yes, 1994
Orbital and pretibial fibroblasts are targets of autoimmune attack in Graves' ophthalmopathy (GO) and pretibial dermopathy (PTD). The fibroblast autoantigen involved in these peripheral manifestations of Graves' disease and the reason for the association
Bahn, Rebecca S.   +3 more
core   +1 more source

Resistance to thyrotropin [PDF]

open access: yesBest Practice & Research Clinical Endocrinology & Metabolism, 2017
Resistance to thyrotropin (RTSH) is broadly defined as reduced sensitivity of thyroid follicle cells to stimulation by biologically active TSH due to genetic defects. Affected individuals have elevated serum TSH in the absence of goiter, with the severity ranging from nongoitrous isolated hyperthyrotropinemia to severe congenital hypothyroidism with ...
Helmut, Grasberger, Samuel, Refetoff
openaire   +2 more sources

Evaluation of the ADVIA (R) Centaur (TM) TSH-3 assay [PDF]

open access: yes, 2000
An analytical evaluation of the thyroid stimulating hormone (TSH-3) assay on the Sayer ADVIA(R) Centaur(TM) immunoassay system was performed. General analytical requirements (linearity, resistance to typical interferences, absence of a carry-over effect)
Bland JM   +6 more
core   +1 more source

Is Thyrotropin Receptor Antibody Positivity Associated with Cytology and Histopathology Results in Patients with Graves’ Disease?

open access: yesEndocrinology Research and Practice, 2022
Objective: The effect of thyrotropin receptor antibody positivity on the cytology and histopathology of nodules developing on the basis of Graves’ disease is unknown.
Hüsniye Başer   +8 more
doaj  

Home - About - Disclaimer - Privacy