Breed- and age-associated variation in hematological parameters and Toll-like receptor 2 gene expression in peripheral blood mononuclear cells of commercial and purebred laying hens during the laying cycle. [PDF]
Rassamepong T +5 more
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Erratum for Petronglo et al., "Context-Dependent Roles for Toll-Like Receptors 2 and 9 in the Pathogenesis of <i>Staphylococcus aureus</i> Osteomyelitis". [PDF]
Petronglo JR +10 more
europepmc +1 more source
Antibody glyco-optimization and site-specific conjugation enhance the immune-stimulating activity of antibody-TLR ligand conjugates. [PDF]
Wang X +6 more
europepmc +1 more source
Extracellular pneumolysin enhances the activation of cytosolic pattern-recognition receptor NOD2 through its pore-forming activity. [PDF]
Domon H +4 more
europepmc +1 more source
Toll like receptor 4 (TLR4) in neuroinflammation: From acute hemorrhagic stroke to chronic neurodegeneration. [PDF]
Choudhary HH +4 more
europepmc +1 more source
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Toll-like Receptor 2 and Toll-like Receptor 4 Expression in Human Adrenals
Hormone and Metabolic Research, 2004Toll-like receptors (TLRs) are key elements in the innate immune response, functioning as pattern-recognition receptors for the detection and response to endotoxins and other microbial ligands. Inflammatory cytokines play an important role in the activation of the hypothalamic-pituitary-adrenal HPA axis during inflammation and sepsis.
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Conditional signaling by Toll‐like receptor 4
The FASEB Journal, 2005ABSTRACT Signaling through Toll‐like receptor 4 (TLR4) is thought to initiate innate and adaptive immune responses. Signaling of TLR4 is usually studied using isolated cells, which are activated by subnanomolar concentrations of lipopolysaccharide (LPS).
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Toll-like receptor-4 and allograft responses
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Toll-like Receptor 4 Polymorphisms and Aspergillosis
New England Journal of Medicine, 2009However, there is another possible explana -tion for the findings that should be considered. During much of the period of the study, trans -plant recipients with febrile neutropenia that was unresponsive to antibiotics received amphotericin B. This is significant because conventional and lipid formulations of amphotericin B activate phagocytic cells by
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