Results 291 to 300 of about 5,576,002 (348)
, 2002 Some of the next articles are maybe not open access.
Related searches:
Related searches:
Microparticles in tumor progression
Thrombosis Research, 2012Microparticles (MP) are shed from the surface of activated or apoptotic blood cells and their levels in plasma reflect a balance between cell stimulation, proliferation, and death. MP production occurs through vesiculation of cell membranes, and involves cytoskeletal changes and a shift in the normal phospholipid asymmetry.
Falanga, A, Tartari, CJ, Marchetti, M
openaire +2 more sources
Science Signaling, 2022
A bacterial metabolite promotes the progression of colorectal cancer.
openaire +2 more sources
A bacterial metabolite promotes the progression of colorectal cancer.
openaire +2 more sources
“Stromatogenesis” and Tumor Progression
International Journal of Surgical Pathology, 2004“Stromatogenesis” is the formation of new stroma occurring, in parallel with the neoplastic process, at sites of active tumor invasion, i.e., at the free surface of a developing exophytic tumor, at the invading tumor front of an advancing endophytic tumor, and at sites of tumor metastasis, wherein the newly formed stroma disrupts the continuity of ...
Efthimios, Sivridis +2 more
openaire +2 more sources
Extracellular matrix stiffness: mechanisms in tumor progression and therapeutic potential in cancer
Experimental Hematology & OncologyTumor microenvironment (TME) is a complex ecosystem composed of both cellular and non-cellular components that surround tumor tissue. The extracellular matrix (ECM) is a key component of the TME, performing multiple essential functions by providing ...
Meiling Zhang, Bin Zhang
semanticscholar +1 more source
Tetraspanins and tumor progression
Clinical & Experimental Metastasis, 2010Transmembrane protein tetraspanins either promote or suppress tumor invasion and metastasis. Their effects on tumor progression depend on the multimolecular transmembrane complex called tetraspanin-enriched microdomain (TEM) and are attributed to the alterations in the (1) motogenic and mitogenic behaviors and/or (2) microenvironmental interactions of ...
Mekel M, Richardson +2 more
openaire +2 more sources
Medical Hypotheses, 2020
A model is introduced here that for the first time describes carcinogenesis in the context of and interacting with associated inflammatory processes. Central to the model are the control of cytokine production by the innate immune system and its disturbance by additional uncontrolled cytokine sources.
openaire +2 more sources
A model is introduced here that for the first time describes carcinogenesis in the context of and interacting with associated inflammatory processes. Central to the model are the control of cytokine production by the innate immune system and its disturbance by additional uncontrolled cytokine sources.
openaire +2 more sources
Neurotrophin receptors, tumor progression and tumor maturation
Molecular Medicine Today, 1996The nerve growth factor receptor TrkA was initially isolated as a transforming oncogene, trk, in which most of the extracellular receptor part is replaced by the coding sequence for a tropomyosin-encoding gene. The impact that the identification of the first neurotrophin receptor has made on the entire field of developmental neurobiology cannot be ...
S, Påhlman, J C, Hoehner
openaire +2 more sources
Intracellular Entropy-driven Multi-bit DNA Computing for Tumor Progression Discrimination.
Angewandte Chemie, 2020Tumor progression such as metastasis are complicated events that involve abnormal expression of different miRNAs and enzymes. Monitoring these biomolecules in live cells with computational DNA nanotechnology may enable discrimination of tumor progression
Min Bai +7 more
semanticscholar +1 more source
Differentiation and Tumor Progression
1995Clinical and experimental experience indicate that differentiation and malignancy are inversely correlated. However, more recent experimental studies using mouse and human keratinocyte systems have demonstrated that complete or even substantial loss in overall epithelial differentiation is not a prerequisite for malignant growth of cancer cells.
N E, Fusenig +4 more
openaire +2 more sources