Results 171 to 180 of about 66,565 (306)

High‐Dose Parenteral Iron Corrects Iron Deficiency Anemia While Disrupting Gut Microbiota and Metabolic Homeostasis in Neonatal Piglets

open access: yesFood Frontiers, Volume 7, Issue 1, January 2026.
Our results indicate that although high‐dose parenteral iron effectively corrects iron deficiency anemia (IDA), it causes liver dysfunction, immune suppression, systemic iron overload, and altered gut microbiota. ABSTRACT Iron deficiency anemia (IDA) is a common nutritional disorder in mammals, particularly affecting neonates and neonatal piglets ...
Qian Zhang   +4 more
wiley   +1 more source

Multi-target antidiabetic therapy with voglibose, ubiquinone, and tempol: synergistic effects on liver and skeletal muscle in experimental type 2 diabetes. [PDF]

open access: yesBMC Pharmacol Toxicol
Akarslan ÖT   +8 more
europepmc   +1 more source

Cytochrome b, flavins, and ubiquinone-50 in enucleated human neutrophils (polymorphonuclear leukocyte cytoplasts).

open access: hybrid, 1984
René Lutter   +4 more
openalex   +1 more source

The subcellular localization of ubiquinone in human neutrophils [PDF]

open access: bronze, 1983
Andrew R. Cross   +3 more
openalex   +1 more source

Functional Screening of NDUFAF6 Variants in Knockout Cells and Complementary Computational Analysis

open access: yesJournal of Clinical Laboratory Analysis, Volume 40, Issue 1, January 2026.
We established an ATP‐based functional screening system in NDUFAF6 knockout cells to evaluate the pathogenicity of gene variants associated with mitochondrial dysfunction. Our results highlight the limitations of in silico predictions and underscore the necessity of experimental validation for accurate variant interpretation.
Feng Jiang   +7 more
wiley   +1 more source

Energy Metabolism Under Stress: Late‐Stage Leigh Syndrome Reveals Profound Cardiometabolic Perturbations in Ndufs4 KO Mice

open access: yesJournal of Inherited Metabolic Disease, Volume 49, Issue 1, January 2026.
Cardiac bioenergetic and metabolic perturbations in late‐stage Leigh syndrome. Whole‐body NDUFS4 loss severely impairs cardiac complex I activity and respiration, shifting reliance to complex II and reducing glycolytic, TCA, and amino acid‐derived energy‐generating substrates in the heart.
Karin Terburgh   +2 more
wiley   +1 more source

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