Results 221 to 230 of about 99,858 (332)
BIOCHEMICAL OBSERVATIONS ON E. COLI MUTANTS DEFECTIVE IN URIDINE DIPHOSPHOGLUCOSE [PDF]
T. A. Sundararajan+2 more
openalex +1 more source
Abstract Objective Mild malformations of cortical development with oligodendroglial hyperplasia in epilepsy (MOGHE) are brain lesions associated with focal epilepsy and characterized by increased oligodendroglial density, heterotopic neurons, and hypomyelination in the white matter.
Isabella C. Galvão+10 more
wiley +1 more source
Status epilepticus in older adults: A critical review
Abstract Older adults (≥60 years of age) have the highest incidence of status epilepticus (SE) among adults and experience the highest morbidity and mortality. SE incidence increases with age in adulthood. A recent study from Austria estimated an incidence of 89.6/100 000 and 67.6/100 000 person‐years adjusted for age and sex in women and men aged >60 ...
Matthew R. Woodward+5 more
wiley +1 more source
Phosphorylation of nucleosides by P-N bond species generated from prebiotic reduced phosphorus sources. [PDF]
Gull M+3 more
europepmc +1 more source
Pharmacogenomics of Human Uridine Diphospho‐Glucuronosyltransferases and Clinical Implications
Chantal Guillemette+2 more
semanticscholar +1 more source
Metabolomic and proteomic stratification of equine osteoarthritis
Abstract Background Equine osteoarthritis (OA) is predominantly diagnosed through clinical examination and radiography, leading to detection only after significant joint pathology. The pathogenesis of OA remains unclear and while many medications modify the disease's inflammatory components, no curative or reversal treatments exist.
James R. Anderson+5 more
wiley +1 more source
Simply cut out - Combining CRISPR/Cas9 RNPs and transiently selected telomere vectors for marker free-gene deletion in <i>Trichoderma atroviride</i>. [PDF]
Gründlinger M+6 more
europepmc +1 more source
Pseudouridine: Still mysterious, but never a fake (uridine)!
Felix Spenkuch, Y. Motorin, M. Helm
semanticscholar +1 more source
Gle1 knockout mice fail to segregate cell lineages at the blastocyst stage, resulting in very early embryonic lethality. Gle1 knock‐in (KI) mice harboring a pathogenic variant giving rise to lethal congenital contracture syndrome 1 show both known and novel innervation defects, supportive of multiorgan pathology in human fetuses.
Tomáš Zárybnický+22 more
wiley +1 more source