Prussian Blue Nanozyme Potentiates Venetoclax Efficacy in Acute Myeloid Leukemia by Inducing a More Mature Phenotype. [PDF]
Yang F +10 more
europepmc +1 more source
Targeting WNK1 suppresses acute myeloid leukemia progression and enhances sensitivity to venetoclax. [PDF]
Chen F +8 more
europepmc +1 more source
Venetoclax retreatment in chronic lymphocytic leukemia is biologically rational and clinically effective. [PDF]
Castonguay M +3 more
europepmc +1 more source
Obinutuzumab, lenalidomide, and venetoclax for the initial treatment of patients with advanced-stage follicular lymphoma: Results of the Phase Ib/II LEVERAGE study. [PDF]
Cheah CY +11 more
europepmc +1 more source
Novel treatment strategies in acute myeloid leukemia. [PDF]
Simkhada S, Joshi U, Dhakal P.
europepmc +1 more source
Real‐world nationwide data in B‐cell malignancies reveal a markedly increased 1‐year incidence of hospitalized atrial fibrillation with ibrutinib compared with second‐generation Bruton's tyrosine kinase inhibitors and venetoclax, arguing for closer cardiac surveillance.
Yoann Zelmat +5 more
wiley +1 more source
EHA Guidelines on management of chronic lymphocytic leukemia and Richter transformation. [PDF]
Eichhorst B +13 more
europepmc +1 more source
Therapeutic targeting of BCL-2 during CART cell production augments potency through non-apoptotic adaptive changes. [PDF]
Aboelella NS +11 more
europepmc +1 more source
MAPK Inhibitor‐Tolerant Persister Cells in Melanoma: Mechanisms and Therapeutic Vulnerabilities
Melanoma response to MAPK inhibition progresses from drug‐sensitive cells to drug‐tolerant persister states, which enable survival through adaptive signaling, metabolic rewiring, and phenotypic plasticity. These persister cells serve as an intermediate reservoir from which resistant tumors ultimately emerge. ABSTRACT Though major advancements have been
Bhoomi Shah +3 more
wiley +1 more source

