Results 121 to 130 of about 15,740 (203)

Prussian Blue Nanozyme Potentiates Venetoclax Efficacy in Acute Myeloid Leukemia by Inducing a More Mature Phenotype. [PDF]

open access: yesInt J Nanomedicine
Yang F   +10 more
europepmc   +1 more source

Targeting WNK1 suppresses acute myeloid leukemia progression and enhances sensitivity to venetoclax. [PDF]

open access: yesFront Oncol
Chen F   +8 more
europepmc   +1 more source

Obinutuzumab, lenalidomide, and venetoclax for the initial treatment of patients with advanced-stage follicular lymphoma: Results of the Phase Ib/II LEVERAGE study. [PDF]

open access: yesHemasphere
Cheah CY   +11 more
europepmc   +1 more source

Comparative atrial fibrillation risk across Bruton's tyrosine kinase inhibitors in B‐cell malignancies: A nationwide population‐based study

open access: yesBritish Journal of Haematology, Volume 209, Issue 2, Page 534-543, August 2026.
Real‐world nationwide data in B‐cell malignancies reveal a markedly increased 1‐year incidence of hospitalized atrial fibrillation with ibrutinib compared with second‐generation Bruton's tyrosine kinase inhibitors and venetoclax, arguing for closer cardiac surveillance.
Yoann Zelmat   +5 more
wiley   +1 more source

EHA Guidelines on management of chronic lymphocytic leukemia and Richter transformation. [PDF]

open access: yesHemasphere
Eichhorst B   +13 more
europepmc   +1 more source

Therapeutic targeting of BCL-2 during CART cell production augments potency through non-apoptotic adaptive changes. [PDF]

open access: yesSignal Transduct Target Ther
Aboelella NS   +11 more
europepmc   +1 more source

MAPK Inhibitor‐Tolerant Persister Cells in Melanoma: Mechanisms and Therapeutic Vulnerabilities

open access: yesCancer Science, Volume 117, Issue 8, Page 2050-2063, August 2026.
Melanoma response to MAPK inhibition progresses from drug‐sensitive cells to drug‐tolerant persister states, which enable survival through adaptive signaling, metabolic rewiring, and phenotypic plasticity. These persister cells serve as an intermediate reservoir from which resistant tumors ultimately emerge. ABSTRACT Though major advancements have been
Bhoomi Shah   +3 more
wiley   +1 more source

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