Results 111 to 120 of about 491,136 (305)

Ratio of Blood Glucose Level Change Measurement for Flap Monitoring

open access: yesPlastic and Reconstructive Surgery, Global Open, 2018
Background:. In a setting of flap congestion, early detection and rapid reexploration are important. Some studies described the efficacy of blood glucose measurement for flap monitoring.1 However, the sensitivity and specificity of this method were not ...
Ryo Karakawa, MD   +3 more
doaj   +1 more source

Extracellular Vesicle Packaged TDP43 Derived From ECs Exacerbates Cigarette Tar‐Related Atherosclerosis Progression via Enhancing Macrophage Extracellular Traps

open access: yesAdvanced Science, EarlyView.
Cigarette tar triggers EC‐derived EVs carrying TDP43, which binds VDAC1 to boost MAMs, mitochondrial Ca2+ overload, and METs formation in macrophages, accelerating smoking‐related AS progression, while retinoic acid binds TDP43 to block this pathogenic cascade.
Xinxin Zhu   +20 more
wiley   +1 more source

Factor XIII Val34Leu is a genetic factor involved in the aetiology of venous thrombosis

open access: yes, 1999
A mutation in the factor XIII gene (FXIII Val34Leu) gene was recently reported to confer protection against myocardial infarction, but its relationship with venous thrombosis is unknown.
Franco, R. F.   +8 more
core   +2 more sources

Incidence of deep venous thrombosis and stratification of risk groups in a university hospital vascular surgery unit

open access: yesJornal Vascular Brasileiro, 2015
BACKGROUND: There is a knowledge gap with relation to the true incidence of deep vein thrombosis among patients undergoing vascular surgery procedures in Brazil. This study is designed to support the implementation of a surveillance system to control the
Alberto Okuhara   +3 more
doaj   +1 more source

Cerebral Venous Sinus Thrombosis

open access: yes, 2020
Dr.
Andrew G. Lee, MD; Jae Eun Lee
core  

Inhibition of MEKK3 Prevents Cerebral Cavernous Malformation Progression Via Restoring Endothelial Integrity and Restricting Microglial Activation

open access: yesAdvanced Science, EarlyView.
In this study, we demonstrate that aberrant MEKK3 signaling in endothelial cells and microglia drives the progression of cerebral cavernous malformations (CCMs). Furthermore, we identify DPDH as a potent small‐molecule MEKK3 inhibitor with substantial therapeutic potential for CCM treatment.
Weiwei Zheng   +17 more
wiley   +1 more source

Metabolic landscape in venous thrombosis: insights into molecular biology and therapeutic implications

open access: yesAnnals of Medicine
The findings of the last decade suggest a complex link between inflammatory cells, coagulation, and the activation of platelets and their synergistic interaction to promote venous thrombosis.
Zheng Cao   +3 more
doaj   +1 more source

RBMS1 Contributes to Early Arteriovenous Fistula Neointimal Remodeling Through Modulation of VSMC Phenotypic Responses

open access: yesAdvanced Science, EarlyView.
Postoperative arteriovenous fistulas exhibit increased RBMS1 in vascular smooth muscle cells (VSMCs). In experimental AVFs, RBMS1 overexpression aggravates, whereas deletion attenuates, neointimal hyperplasia. RBMS1 binds MPZL1 mRNA and promotes exon 5 skipping to generate MPZL1‐Δ5, which interacts with G3BP1 to activate AKT, driving VSMC proliferation,
Zongda Li   +14 more
wiley   +1 more source

蛋白C缺乏及蛋白S缺乏是台灣中國人靜脈栓塞好發症最重要之危險因子

open access: yes, 2009
The relative risks (odds ratio, OR) of various risk factors for venous thrombophilia, including sex, antithrombin III, protein C (PC), protein S (PS) and plasminogen deficiencies, and C677T homozygous mutation of methylenetetrahydrofolate reductase gene ...
沈銘鏡;林正修;蔡偉   +1 more
core  

The JAK2 V617F mutation in patients with cerebral venous thrombosis.

open access: yes, 2012
Background: It is currently unclear whether or not cerebral venous thrombosis, such as splanchnic venous thrombosis, can be the first manifestation of an underlying myeloproliferative neoplasm.
Biguzzi E   +8 more
core   +1 more source

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