RT promotes VG161 replication in BC and its immunostimulatory transgenes expression, which is mediated by the upregulation of GADD34 and HVEM caused by RT. The combination therapy with VG161 and RT increases the abundances of tumor‐infiltrating lymphocytes and elicits potent systemic antitumor immunity, thereby effectively inhibiting local tumors and ...
Lijuan Lyu +13 more
wiley +1 more source
Immune Cells at the Frontline of SFTSV Infection. [PDF]
Park S, Kim C.
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The origins and potential cross-species transmission of paramyxoviruses and other RNA viruses between native snakes and invasive Burmese pythons in the Florida Everglades. [PDF]
Shrestha A +12 more
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Sorafenib-associated Trichodysplasia Spinulosa versus Follicular Hyperkeratosis: a case report. [PDF]
Hasan R +4 more
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Rat polyomavirus 2 infection: secondary publication. [PDF]
Tanaka M.
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Liquid-Liquid Phase Separation in Viral Infection and Immunology. [PDF]
Xu J +5 more
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IQGAP1 and IQGAP3 are critical host factors for Marburg virus replication, nucleocapsid transport, and cell-to-cell spread. [PDF]
Dolnik O +8 more
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Viral Inclusion Bodies in Tracheobronchial Epithelium of Asymptomatic Subjects [PDF]
During a survey conducted for the cytodiagnosis of early bronchogenic carcinoma, cytoplasmic viral inclusion bodies were found sporadically in tracheobronchial smears of asymtomatic patients of both sexes (ages ranging from 18 to 80 years) undergoing general endotracheal anesthesia for surgery.
Herman Turndorf
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Disruption of Ebola NP0VP35 Inclusion Body-like Structures reduce Viral Infection
Journal of Molecular Biology, 2023Viral inclusion bodies (IBs) are potential sites of viral replication and assembly. How viral IBs form remains poorly defined. Here we describe a combined biophysical and cellular approach to identify the components necessary for IB formation during Ebola virus (EBOV) infection. We find that the eNP0VP35 complex containing Ebola nucleoprotein (eNP) and
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