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TRIM18 is a critical regulator of viral myocarditis and organ inflammation [PDF]

open access: diamondJournal of Biomedical Science, 2022
Background Infections by viruses including severe acute respiratory syndrome coronavirus 2 could cause organ inflammations such as myocarditis, pneumonia and encephalitis.
Mingli Fang   +9 more
doaj   +3 more sources

Immune mechanisms of group B coxsackievirus induced viral myocarditis

open access: yesVirulence, 2023
Viral myocarditis is known to be a primary cause of dilated cardiomyopathy (DCM) that can lead to heart failure and sudden cardiac death and is invariably caused by myocardial viral infection following active inflammatory destruction of the myocardium ...
Yue Zhang   +4 more
doaj   +2 more sources

A Review of the Role of Imaging Modalities in the Evaluation of Viral Myocarditis with a Special Focus on COVID-19-Related Myocarditis

open access: yesDiagnostics, 2022
Viral myocarditis is inflammation of the myocardium secondary to viral infection. The clinical presentation of viral myocarditis is very heterogeneous and can range from nonspecific symptoms of malaise and fatigue in subclinical disease to a more florid ...
Adedayo Adeboye   +4 more
doaj   +2 more sources

Mechanisms and Therapeutic Strategies of Viral Myocarditis Targeting Autophagy

open access: yesFrontiers in Pharmacology, 2022
Viral myocarditis is caused by infection with viruses or bacteria, including coxsackievirus B3 (CVB3), and is characterized by acute or chronic inflammatory responses in the heart.
Kun Yu   +11 more
doaj   +2 more sources

Viral Myocarditis. [PDF]

open access: yesYale J Biol Med
Viral myocarditis is associated with the development of dilated cardiomyopathy (DCM), left ventricular dysfunction, and heart failure. This review addresses the mechanisms of viral myocarditis and its treatment.
Ying C.
europepmc   +3 more sources

Advances in cell death mechanisms involved in viral myocarditis

open access: yesFrontiers in Cardiovascular Medicine, 2022
Viral myocarditis is an acute inflammatory disease of the myocardium. Although many etiopathogenic factors exist, coxsackievirus B3 is a the leading cause of viral myocarditis.
Yang Yang   +6 more
doaj   +2 more sources

Comparison of Multisystem Inflammatory Syndrome in Children–Related Myocarditis, Classic Viral Myocarditis, and COVID‐19 Vaccine‐Related Myocarditis in Children

open access: yesJournal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, 2022
Background Although rare, classic viral myocarditis in the pediatric population is a disease that carries significant morbidity and mortality. Since 2020, myocarditis has been a common component of multisystem inflammatory syndrome in children (MIS‐C ...
Trisha Patel   +6 more
doaj   +2 more sources

An overview of the immune mechanisms of viral myocarditis [PDF]

open access: bronzeReviews in Medical Virology, 2020
Viral myocarditis has been identified as a major cause of dilated cardiomyopathy (DCM) that can lead to heart failure. Historically, Coxsackieviruses and adenoviruses have been commonly suspected in myocarditis/DCM patients in North America and Europe ...
Ninaad Lasrado, Jay Reddy
openalex   +2 more sources

Loss of TRIM29 mitigates viral myocarditis by attenuating PERK-driven ER stress response in male mice

open access: yesNature Communications
Viral myocarditis, an inflammatory disease of the myocardium, is a significant cause of sudden death in children and young adults. The current coronavirus disease 19 pandemic emphasizes the need to understand the pathogenesis mechanisms and potential ...
Junying Wang   +8 more
doaj   +2 more sources

RETRACTED: MicroRNA‐425‐3p inhibits myocardial inflammation and cardiomyocyte apoptosis in mice with viral myocarditis through targeting TGF‐β1

open access: yesImmunity, Inflammation and Disease, 2021
Objective Emerging articles have profiled the relations between microRNAs and viral myocarditis. This research was unearthed to explore the capacity of miR‐425‐3p on cardiomyocyte apoptosis in mice with viral myocarditis and its mechanism.
Junhua Li, Jiehong Tu, Hong Gao, Lu Tang
doaj   +2 more sources

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