Recognition and research identity in nursing: naming a pattern we have not yet examined. [PDF]
Kelly L.
europepmc +1 more source
Partial inhibition of focal adhesion kinase (FAK) can paradoxically promote tumor growth, rather than simply producing a weaker antitumor effect than that observed with strong FAK suppression. In breast cancer and melanoma models, targeting p110δ PI3K, particularly in macrophages, counteracted these tumor‐promoting effects, highlighting the importance ...
Lydia Xenou +4 more
wiley +1 more source
The Future of Clinical Pharmacology: The Right Medicine at the Right Dose for Each Patient. [PDF]
Peck RW.
europepmc +1 more source
Unraveling the epigenetic code in cancer cell–tumor microenvironment crosstalk
Epigenetic regulation is a key driver of cancer development and progression. Diverse epigenetic alterations in cancer cells and components of the tumor microenvironment (TME) orchestrate their communication through multiple mechanisms. We discuss how the epigenetic code coordinates bidirectional cancer cell–TME crosstalk to promote cancer progression ...
Ji Hoon Park, Mi‐Young Kim
wiley +1 more source
The changing landscape of discipline in Egypt: a descriptive and predictive study across two generations. [PDF]
Zaki NM, Shehata MA, Nasr JA.
europepmc +1 more source
Arginine methylation can be viewed as a persistence‐prone post‐translational modification regulated by a network of PRMTs. Competitive and compensatory interactions among PRMTs can redistribute methylation across substrate pools shaped by sequence, structural, spatial, and environmental layers, reinforcing RNA‐processing, chromatin, and signaling ...
So Hyun Kwon, Ji Min Lee
wiley +1 more source
Relationship between Myers-Briggs Type Indicator, Achievement Motivation, and Clinical Performance of Pharmacy Students in Iran: A Cross-sectional Study. [PDF]
Salarian S +3 more
europepmc +1 more source
Mechanisms and therapeutic opportunities of the ribotoxic stress response in cancer
Cancer cells' high translational demand creates opportunities to therapeutically target ribosome function. Ribosome stalling and collisions activate ZAKα and the ribotoxic stress response (RSR), which can trigger rapid, p53‐independent apoptosis in cancer.
Anastassiya Kim +7 more
wiley +1 more source
One-minute discipline classroom management strategies that work
Short, Jeffrey, Bianco, Arnie
core
From Operating System to Operating Society: The Next Mission of Medical Informatics. [PDF]
Lee KH, Baik S.
europepmc +1 more source

