镉暴露对肾细胞凋亡、自噬、氧化损伤及TGF-β1/Smad3/miRNAs信号通路的影响
目的 探讨镉(Cadmium, Cd)对人胚胎肾细胞293[human embryonic kidney(HEK) 293 cell line, HEK293]细胞凋亡、自噬、氧化损伤以及转化生长因子-β1(Transforming Growth Factor-beta 1,TGF-β1)/Smad同源物3(SMAD3)重组蛋白(Recombinant SMAD family member 3,Smad3)/微核糖核酸(microRNAs, miRNAs)信号通路的影响。方法 用氯化镉(Cadmium ...
刘仁洪, 黄月明, 樊婵, 易宗娓
doaj
HPCAL1 is a novel driver of autophagy-dependent ferroptosis. [PDF]
Wang L, Li Q, Liu H, Li L.
europepmc +1 more source
[Silybin inhibits fibrosis after glaucoma filtration surgery in rabbits by promoting fibroblast autophagy]. [PDF]
Zhang Y +5 more
europepmc +1 more source
[Ophiopogonin D alleviates doxorubicin-induced myocardial hypertrophy in mice by activating the β-catenin/FUNDC1/mitophagy axis]. [PDF]
Lei Y, Song J, Xu L, Liu R, Zhao Y.
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Progress in the effect of microRNA-21 on diseases via autophagy. [PDF]
Zeng Z, Cai J, Liao Y, Sun S, Xie L.
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nIL-33介导的巨噬细胞极化在口腔鳞状细胞癌中的作用及机制研究进展
口腔鳞状细胞癌(OSCC)是世界范围内最常见的头颈部恶性肿瘤,OSCC的发生与口腔黏膜的癌前病变密切相关。巨噬细胞是黏膜免疫的重要参与者,通过调控不同表型的极化状态促进OSCC进展。核白细胞介素-33(nIL-33)通过调控细胞线粒体代谢重塑巨噬细胞极化。本文系统综述巨噬细胞与口腔鳞癌进展的关系,从细胞核-线粒体调控极化表型角度阐明巨噬细胞极化自我调控机制,以期为靶向巨噬细胞抗OSCC治疗提供思路和参考。
王娜 +5 more
doaj
Energy deprivation-induced autophagy and aggrephagy: insights from yeast and mammals. [PDF]
Fan S, Chen Y, Yao W, Yi C.
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Regulation of pyroptosis and ferroptosis by mitophagy in chronic kidney disease. [PDF]
Huang Y, Yang L.
europepmc +1 more source
Vitamin D/vitamin D receptor, autophagy, and infection. [PDF]
Hou Y, Li J, Deng C.
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Autophagy receptor-inspired chimeras: a novel approach to facilitate the removal of protein aggregates and organelle by autophagy degradation. [PDF]
Wang L, Liu H, Li L.
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