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Annals of Clinical Biochemistry: International Journal of Laboratory Medicine, 1989
About ten years ago, Zilva' argued that there is a fundamental difference between hydrolysis of adenosine triphosphate (ATP) under aerobic and anaerobic conditions: 'H+ produced by hydrolysis of ATp4yielded by anaerobic glycolysis is only significant because it is not used in resynthesis' (of ATP presumably).
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About ten years ago, Zilva' argued that there is a fundamental difference between hydrolysis of adenosine triphosphate (ATP) under aerobic and anaerobic conditions: 'H+ produced by hydrolysis of ATp4yielded by anaerobic glycolysis is only significant because it is not used in resynthesis' (of ATP presumably).
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Annals of Internal Medicine, 1970
Excerpt To the editor: A case of lactic acidosis is described in the September issue (OLIVAPP, SCHWARTZHA: Survival of a patient with spontaneous lactic acidosis).
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Excerpt To the editor: A case of lactic acidosis is described in the September issue (OLIVAPP, SCHWARTZHA: Survival of a patient with spontaneous lactic acidosis).
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Nutrition in Clinical Practice, 2005
d‐Lactic acidosis, also referred to as d‐lactate encephalopathy, is a rare neurologic syndrome that occurs in individuals with short bowel syndrome or following jejuno‐ileal bypass surgery. Symptoms typically present after the ingestion of high‐carbohydrate feedings.
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d‐Lactic acidosis, also referred to as d‐lactate encephalopathy, is a rare neurologic syndrome that occurs in individuals with short bowel syndrome or following jejuno‐ileal bypass surgery. Symptoms typically present after the ingestion of high‐carbohydrate feedings.
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Orvosi hetilap, 1991
The rare congenital lactic acidosis is a consequence of enzyme defects. The acquired form is relatively common in critically ill patients. The altered metabolism of pyruvate and the imbalance between lactate production and utilization have a central role in the pathogenesis of this disease.
J, Winkler, G, Boner
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The rare congenital lactic acidosis is a consequence of enzyme defects. The acquired form is relatively common in critically ill patients. The altered metabolism of pyruvate and the imbalance between lactate production and utilization have a central role in the pathogenesis of this disease.
J, Winkler, G, Boner
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