Results 161 to 170 of about 22,051 (209)

Identification of Endogenous Adenomatous Polyposis Coli Interaction Partners and β-Catenin-Independent Targets by Proteomics. [PDF]

Mol Cancer Res, 2019
Popow O, Paulo JA, Tatham MH, Volk MS, Rojas-Fernandez A, Loyer N, Newton IP, Januschke J, Haigis KM, Näthke I.   +9 more
europepmc   +1 more source

Truncated Adenomatous Polyposis Coli Mutation Induces Asef-Activated Golgi Fragmentation. [PDF]

Mol Cell Biol, 2018
Kim SB, Zhang L, Yoon J, Lee J, Min J, Li W, Grishin NV, Moon YA, Wright WE, Shay JW.   +9 more
europepmc   +1 more source

Familial Adenomatous Polyposis-Risk of Cancer, Cancer Prevention, and Long-Term Consequences: Learnings from Five Decades with the Danish Polyposis Register. [PDF]

APMIS
Karstensen JG.
europepmc   +1 more source

Identification of an Adenomatous Polyposis Coli Mutation Associated with Attenuated Familial Adenomatous Polyposis

Journal of the Advanced Practitioner in Oncology, 2010
openaire   +1 more source

Genotype-Phenotype Correlations in Attenuated Adenomatous Polyposis Coli [PDF]

American Journal of Human Genetics, 1998
Germ-line mutations of the tumor suppressor APC are implicated in attenuated adenomatous polyposis coli (AAPC), a variant of familial adenomatous polyposis (FAP). AAPC is recognized by the occurrence of 40 years). The aim of this study was to assess genotype-phenotype correlations in AAPC families.
C Soravia, Terri Berk, Lisa Madlensky
exaly   +5 more sources

Mutation analysis of the adenomatous polyposis coli (APC) gene in Danish patients with familial adenomatous polyposis (FAP)

Human Mutation, 2004
Development of one hundred or more adenomas in the colon and rectum is diagnostic for the dominantly inherited, autosomal disease Familial Adenomatous Polyposis (FAP).
Marie Luise Bisgaard, Rasmus S Ripa, S Bülow   +2 more
exaly   +1 more source

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Profuse familial adenomatous polyposis with an Adenomatous Polyposis Coli exon 3 mutation

Familial Cancer, 2001
The attenuated form of familial adenomatous polyposis coli (AAPC) is associated with mutations in the adenomatous polyposis coli (APC) gene which cluster in the 5' region of the gene. It has been proposed that a 'genotype-phenotype boundary' exists at codons 159-163, and mutations that are 5' of this boundary will produce AAPC.
S, Nasioulas, I T, Jones, D J, St John, R J, Scott, S M, Forrest, R J, McKinlay Gardner   +5 more
openaire   +2 more sources

Adenomatous polyposis coli proteins and cell adhesion

Current Opinion in Cell Biology, 2004
Adenomatous polyposis coli (APC) is an important tumour suppressor in the mammalian intestinal epithelium. It binds to beta-catenin and its role as a tumour suppressor depends predominantly on its ability to downregulate soluble beta-catenin, a key effector of the Wnt signalling pathway.
Mariann Bienz, Fumihiko Hamada
exaly   +3 more sources

Adenomatous polyposis coli and translational medicine

The Lancet, 1996
See page 433 Advances in molecular biological techniques in recent years have greatly increased our level of understanding about the mechanisms underlying carcinogenesis. Such advances have also enabled identification of genetic mutations associated with specific cancers.
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Adenomatous polyposis coli gene large deletions in Iranian patients with familial adenomatous polyposis

Indian Journal of Cancer, 2014
Context: Familial adenomatous polyposis (FAP) is one type of hereditary colon cancer with a large number of precancerous polyps that initiation to growth in childhood and adolescent. Mutation in adenomatous polyposis coli (APC) gene is the cause of FAP.
R, Kishani Farahani, M, Montazer Haghighi, H, Asadzadeh Aghdaei, F, Keshavarzi, M Y, Taleghani, F, Goodarzi, E, Nazemalhosseini-Mojarad, M R, Zali   +7 more
openaire   +2 more sources