Results 301 to 310 of about 160,546 (318)
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The effect of glucocorticoids on adipocyte corticotropin receptors and adipocyte responses

Biochimica et Biophysica Acta (BBA) - General Subjects, 1981
A specific and sensitive assay for determining the binding of adrenocorticotropin (ACTH) to isolated rat adipocytes has been developed and utilized to study the effect of glucocorticoids on ACTH receptor. Measurement of the binding of tritiated ACTH (spec. act.
Catherine Behrens, J. Ramachandran
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Adipocyte and Chemokines: A Link between Preadipocyte/Adipocyte and Macrophage in Adipocyte- Related Pathologies

Preventive Nutrition and Food Science, 2004
This review will present a brief overview on the adipocytokines and chemokines in terms of their classifications and functions, and further discuss the most recent results of chemokine research into their regulation of adipocyte functions and/or adipocyte-related pathologies. The potential link between preadipocytes/adipocytes and macrophages will also
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Adipocytes in the Tumour Microenvironment

2020
Adipose tissue contribution to body mass ranges from 6% in male athletes to over 25% in obese men and over 30% in obese women. Crosstalk between adipocytes and cancer cells that exist in close proximity can lead to changes in the function and phenotype of both cell types. These interactions actively alter the tumour microenvironment (TME).
Nikitha K Pallegar, Sherri L. Christian
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Regulation of Adipocyte Development

Annual Review of Nutrition, 1994
INTRODUCTION . . . • . . . . . . • . . . . . . . . . . . . . . . . . . . . . . . . . • • . . . . . . . . • . • . . . . . . • 99 CELL CULTURE MODELS FOR ADIPOCYTE DEVELOPMENT . • • . . . . . . • . • . . . . 101 Multipotent Stem-Cell Lines . • • • . • . • • • • • . • . • . • • • • • • . • • • • • • • • • . • . • • • • • . • • .
M D Lane   +2 more
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Adipocyte hormones

2016
Leptin, adiponectin, acylation stimulating protein, and resistin are called adipokines, which are produced and secreted from adipocytes and have various physiological functions. Leptin is secreted from white adipocytes and suppresses appetite by acting on the hypothalamus.
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[Leptin: adipocyte hormone].

Recenti progressi in medicina, 1998
The authors reviewed the most recent literature on leptin, a protein produced by adipocytes which exerts its action on hypothalamus, modifying eating behavior and inhibiting the lust for food consumption. This one appeared to be the main, if not the only, physiologic action of leptin.
CASTAGNA L   +8 more
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Insulin signaling in the adipocyte

International Journal of Obesity, 2000
Mammalian adipose tissue serves a number of functions, including storage of nutrients for periods of fasting and control of organismal metabolism. Critical to these functions is the capacity of the fat cell to respond to insulin with a significant increase in glucose uptake.
Morris J. Birnbaum   +2 more
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The Adipocyte-Number Hypothesis

Child Development, 1981
The adipocyte-number hypothesis was derived from experimental studies of rats. It states the number of adipocytes (fat cells) is fixed early in life and predestines an individual to be lean or obese depending on changes in the number of adipocytes. However, the present methods for estimating adipocyte number are based on the estimation of total body ...
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Adipocyte–Brain: Crosstalk

2010
The initial discovery of leptin, an appetite-suppressing hormone originating from fat tissue, substantially supported the idea that fat-borne factors act on the brain to regulate food intake and energy expenditure. Since then, a growing number of cytokines have been found to be released from adipose tissue, thus acting in an endocrine manner.
Hendrik Lehnert   +2 more
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The adipocyte as an endocrine cell1

Journal of Animal Science, 2004
Communication between adipose and other tissues has been hypothesized since at least the 1940s to be bidirectional. Despite this expectation, early progress was largely limited to adipose tissue's role in metabolism and storage of fatty acids, its development, and its response to endocrine and neural cues.
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