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Abstract The phosphodiesterase‐5 inhibitor sildenafil suppresses ventricular arrhythmias in a sheep model of drug‐induced long QT. In that study, ventricular arrhythmias were abolished by reducing premature ventricular complexes (PVCs) and delaying PVC onset, thus preventing ‘R‐on‐T’ ventricular tachycardia.
David C. Hutchings +3 more
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Drug Repositioning in Doxorubicin-Induced Cardiotoxicity Protection. [PDF]
Kosić M +3 more
europepmc +1 more source
Effects of Skeletal Muscle Hypertrophy on Fat Mass and Glucose Homeostasis in Humans and Animals: A Narrative Review with Systematic Literature Search. [PDF]
Havers T +4 more
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Neurotransmitters and Immunity: Molecular Mechanisms, Biological Functions, Diseases, and Potential Therapeutic Targets. [PDF]
Li G +13 more
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Microbiota-derived aromatic amino acid decarboxylases: linking microbial fitness and host neurochemical communication. [PDF]
Rahmdel S, Luqman A, Götz F.
europepmc +1 more source
Beyond the heart: a review exploring non-cardiovascular effects of vasoactive agents. [PDF]
Lan P +5 more
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The crosstalk between nerves and immunity: chronic stress as a driver of tumor progression. [PDF]
He Y +7 more
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Developmental Biology, 1989
Tricyclic antidepressants in combination with in vitro clonal analysis of quail neural crest cells were used to examine the role the norepinephrine uptake mechanism might play in the development of adrenergic neural crest derivatives. Norepinephrine (NE) uptake inhibitors blocked expression of the adrenergic phenotype by neural crest cells.
Maya Sieber-Blum
openaire +3 more sources
Tricyclic antidepressants in combination with in vitro clonal analysis of quail neural crest cells were used to examine the role the norepinephrine uptake mechanism might play in the development of adrenergic neural crest derivatives. Norepinephrine (NE) uptake inhibitors blocked expression of the adrenergic phenotype by neural crest cells.
Maya Sieber-Blum
openaire +3 more sources

