Results 131 to 140 of about 532,309 (312)
Cancer Management and Research, 2020 Lanxiang Bai1 ,* Xiaonu Peng2 ,* Ruimei Sun3 1Disinfection Supply Center, Yantai Yuhuangding Hospital, Yantai 264000, Shandong, People’s Republic of China; 2Department of Thoracic Surgery, Yantai Yuhuangding Hospital, Yantai 264000, Shandong ...
Bai L, Peng X, Sun R
doaj
Advanced Materials, EarlyView.This project developed a smart bandage‐like patch (a microneedle array) for repairing diabetic bone damage. It intelligently senses signals from infection and inflammation, then releases its medicines in a specific, timed sequence: first an antibacterial agent, then an anti‐inflammatory agent, and finally growth factors.
Yu Wang +10 more
wiley +1 more source
Siglec-E promotes β2-integrin-dependent NADPH oxidase activation to suppress neutrophil recruitment to the lung [PDF]
, 2014 Siglec-E is a sialic acid-binding Ig-like lectin expressed on murine myeloid cells. It has recently been shown to function as a negative regulator of ß2-integrin-dependent neutrophil recruitment to the lung following exposure to lipopolysaccharide (LPS).
Hegde, Vikas +9 more
core +1 more source
Neuropsychiatric Disease and Treatment, 2021 Wei Zeng,1,2 Chunyuan Zhang,2 Qingshan Long,3 Yalan Li1 1Department of Anesthesiology, The First Affiliated Hospital of Jinan University, Guangzhou, 510630, Guangdong, People’s Republic of China; 2Department of Anesthesiology, Affiliated Boai ...
Zeng W, Zhang C, Long Q, Li Y
doaj
Chromosomal Instability Drives Glioblastoma Heterogeneity and Therapeutic Opportunities
Advanced Science, EarlyView.ABSTRACT Glioblastoma, the most aggressive and lethal form of brain cancer, is defined by profound genomic instability, with Chromosomal Instability (CIN) playing a central role in driving tumor progression, therapy resistance, and poor prognosis. CIN is characterized by numerical and structural alterations, is driven by mechanisms such as mitotic ...
Amarnath Pal +3 more
wiley +1 more source
, 2018 Figure S2 Total AKT level is not changed in Mig-6 d/d mice after P4 treatment. (A) The expression of AKT in the uteri of vehicle (a) and P4 (b) treated Mig-6 d/d mice and (B) H-score of AKT in the uteri of vehicle and P4 treated Mig-6 d/d mice. (PPTX 407
Jung-Yoon Yoo (462528) +5 more
core +1 more source
PD‐1 Inhibits CD4+ TRM‐Mediated cDC1 Mobilization via Suppressing JAML in Human NSCLC
Advanced Science, EarlyView.CD4+ tissue‐resident memory T cells (TRMs) in non‐small cell lung cancer recruit conventional type 1 dendritic cells via XCL1‐XCR1 signaling, orchestrating antitumor immunity. The costimulatory molecule JAML is essential for this process. PD‐1 blockade restores JAML expression and cDC1 mobilization, while JAML agonists synergize with anti‐PD‐1 therapy,
Zheyu Shao +16 more
wiley +1 more source
, 2018 Figure S1 Total AKT level is not changed in Mig-6 f/f and Mig-6 d/d mice. (A) The expression of AKT in the uteri of Mig-6 f/f (a) and Mig-6 d/d (b) mice and (B) H-score of AKT in the uteri of Mig-6 f/f and Mig-6 d/d mice.
Jung-Yoon Yoo (462528) +5 more
core +1 more source
Advanced Science, EarlyView.Malectin alleviates high glucose‐induced ER stress and damage in placental trophoblasts, a function dependent on its six critical carbohydrate‐binding residues. In a GDM mouse model, administration of TAT‐Malectin ameliorated hyperglycemia and placental ER stress and prevented fetal macrosomia.
Jiahui Zhu +12 more
wiley +1 more source
Figure 8: NDNF Rejuvenated Old hBM-MSCs and Improved Cardiac Function Through Activation of p-Akt
, 2019 (A) The phosphorylated Akt (p-Akt) level in old NDNF-transduced hBM-MSCs (old+NDNF) was significantly higher than in empty vector–transduced cells (old). (B) Western blotting showed the expression of p-Akt protein in the border area of mouse heart tissue
Ren-Ke Li (567108) +13 more
core +1 more source