Results 221 to 230 of about 50,798 (264)
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Mediators and Allergic Inflammation of Human Airways

Respiration, 2009
Local antigen challenge in patients with respiratory allergy is associated with histamine and arachidonic acid metabolites release, both in upper and in lower respiratory airways. Raised histamine levels can be detected in nasal secretions 5 min after allergen stimulation.
A. Miadonna   +7 more
openaire   +3 more sources

Hemopoietic Mechanisms in Allergic Airway Inflammation

International Archives of Allergy and Immunology, 1998
The bone marrow actively participates in the production of IgER–positive inflammatory cells (eosinophils, basophils and mast cells), which are typically recruited to tissues in atopic individuals. Understanding the signalling between the tissue and the bone marrow at the molecular level may well open up new avenues of therapy for allergic inflammation.
J A, Denburg   +4 more
openaire   +2 more sources

Interleukin 25 in Allergic Airway Inflammation

International Archives of Allergy and Immunology, 2006
T helper 2 (Th2) cells induce allergic inflammation through the production of cytokines such as interleukin (IL)-4, IL-5 and IL-13. Recently, it has been demonstrated that a novel IL-17 family cytokine IL-25 (IL-17E) is a product of activated Th2 cells and mast cells.
Tomohiro, Tamachi   +4 more
openaire   +2 more sources

Allergic airway inflammation in hypothyroid rats

Journal of Allergy and Clinical Immunology, 1999
Hormones play a modulating role in allergic inflammation. Hyperthyroidism may increase the severity of asthma, and hypothyroidism may ameliorate coexistent asthma. The mechanisms regulating this interaction are not completely understood.The purpose of this study was to test the hypothesis that thyroid hormones influence the development of allergic ...
S, Manzolli   +3 more
openaire   +2 more sources

Role of platelets in allergic airway inflammation

Journal of Allergy and Clinical Immunology, 2015
Increasing evidence suggests an important role for platelets and their products (e.g., platelet factor 4, β-thromboglobulin, RANTES, thromboxane, or serotonin) in the pathogenesis of allergic diseases. A variety of changes in platelet function have been observed in patients with asthma, such as alterations in platelet secretion, expression of surface ...
Idzko, Marco   +2 more
openaire   +3 more sources

Leukocyte Infiltration in Allergic Airway Inflammation

American Journal of Respiratory Cell and Molecular Biology, 1995
Abstract The pathologic manifestation of an allergic airway response is a culmination of several coordinated events involving the participation of multiple mechanisms and cell types. The early release of mast cell-derived arachidonic acid metabolites, production of early-response cytokines, and increased expression of adhesion ...
N W, Lukacs, R M, Strieter, S L, Kunkel
openaire   +2 more sources

Peanut allergy and allergic airways inflammation

Pediatric Allergy and Immunology, 2010
Hughes JL, Brown T, Edgar JD, Shields MD. Peanut allergy and allergic airways inflammation.
Pediatr Allergy Immunol 2010: 21: 1107–1113.
© 2010 John Wiley & Sons A/SAsthma is a major risk cofactor for anaphylactic deaths in children with peanut allergy.
Hughes, Jennifer L.   +3 more
openaire   +3 more sources

Dissociation between Airway Inflammation and Airway Hyperresponsiveness in Allergic Asthma

American Journal of Respiratory and Critical Care Medicine, 1998
Abstract In asthma, the acute increment of airway responsiveness caused by exposure to allergen is associated with influx of eosinophils into the airways. The relationship between chronic airway hyperresponsiveness and airway inflammation is unclear, as they do not change consistently following long-term anti-inflammatory treatments ...
CRIMI, EMANUELE   +5 more
openaire   +4 more sources

Allergic inflammation in upper and lower airways

Annals of Allergy, Asthma & Immunology, 1999
The primary reason for this review is to discuss the relationship between upper and lower airways at various levels with the emphasis on common pathophysiologic mechanisms, and how treatment of the upper airways will benefit the lower airways.The main source of information is derived from original articles and books, with an extensive bibliography ...
openaire   +2 more sources

Effect of IL-37 on Allergic Airway Inflammation

Annals of the American Thoracic Society, 2016
Abstract Rationale The new cytokine IL-37 has been described as a negative regulator of innate immunity. It reduces activation of dendritic cells and the production of proinflammatory mediators in murine and human immune cells.
Lars, Lunding   +7 more
openaire   +2 more sources

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