Results 11 to 20 of about 35,852 (230)

Alopecia areata. Diagnostic and therapeutic recommendations of the Polish Society of Dermatology. Part 2: Treatment

open access: yesPrzegląd Dermatologiczny, 2023
The treatment goal in alopecia areata is induction of hair regrowth and halting the progression of the disease. Treatment decisions depend on the severity of the alopecia areata as assessed by the Severity of Alopecia Tool (SALT) or the Alopecia Areata ...
Adriana Rakowska   +8 more
doaj   +2 more sources

Alopecia areata. Diagnostic and therapeutic recommendations of the Polish Dermatological Society. Part 1. Diagnosis and severity assessment

open access: yesPrzegląd Dermatologiczny, 2023
The existence of two types of alopecia areata is suggested (probably genetically different): early-onset, with more severe symptoms, longer disease duration, higher tendency to relapse, often with a positive family history, and late-onset, in which the ...
Adriana Rakowska   +8 more
doaj   +2 more sources

Progress in immune pathogenesis and targeted therapy of alopecia areata

open access: yesPifu-xingbing zhenliaoxue zazhi, 2023
Alopecia areata is a common clinical non-cicatricial alopecia. Alopecia totalis and generalized alopecia may occur in severe cases. The pathogenesis of alopecia areata is complex, and the treatment is faced with many difficulties.
Jiali WANG   +3 more
doaj   +2 more sources

Evidence Based Treatment of Alopecia Areata [PDF]

open access: yesBerkala Ilmu Kesehatan Kulit dan Kelamin (Periodical of Dermatology and Venerology), 2017
Background: Alopecia areata is a chronic autoimmune disease, involving non-scarring hair loss, which affects hair follicles and sometimes nails. Hair loss pattern presents as patchy alopecia, ophiasis, ophiasis inversa (sisapho), reticularis or diffuse ...
Eva Hariani, Nelva K. Jusuf
doaj   +3 more sources

Tofacitinib in a Recalcitrant Case of Alopecia Areata [PDF]

open access: yesGAIMS Journal of Medical Sciences, 2022
Alopecia areata (AA) is a non-cicatricial alopecia that is postulated to be a hair-specific autoimmune disease, with genetic factors playing a role in disease susceptibility and severity.
Mansi Pithadia   +3 more
doaj   +2 more sources

Alopecia Areata

open access: yesArchives of Dermatology, 1985
TO THE EDITOR.— Alopecia areata (AA) is a common dermatologic disease, the origin of which is still unknown.1 A number of studies support the hypothesis that at least a subset of alopecia areata may be immunologically mediated.
D, Zauli   +5 more
core   +5 more sources

CORTICOSTEROID AS TREATMENT OF ALOPECIA AREATA

open access: yese-Jurnal Medika Udayana, 2013
Alopecia areata is a common cause of alopecia nonscarring. Incidence alopecia areata 1-2% of the population, with the same ratio in both sexes, and occurs in all racial groups.
Ni Putu Junika Putri, I Wayan Sugiritama
doaj   +1 more source

Alopecia Areata Associated with Localized Vitiligo

open access: yesCase Reports in Dermatology, 2010
Alopecia areata is a common cause of noncicatricial alopecia that occurs in a patchy, confluent or diffuse pattern. It may occur as a single, self-limiting episode or may recur at varying intervals over many years. The association of alopecia areata with
Shankarling D. Kuchabal, D.S. Kuchabal
doaj   +2 more sources

Treatment of Refractory Segmental Vitiligo and Alopecia Areata in a Child with Upadacitinib and NB-UVB: A Case Report [PDF]

open access: yesClinical, Cosmetic and Investigational Dermatology
Yuzhu Mu, Ting Pan, Lingling Chen Department of Dermatology, The Affiliated Suzhou Hospital of Nanjing Medical University, Suzhou Municipal Hospital, Suzhou, Jiangsu, 215000, People’s Republic of ChinaCorrespondence: Lingling Chen, Email chenlingling031 ...
Mu Y, Pan T, Chen L
doaj   +1 more source

Alopecia areata and autoimmunity: A clinical study

open access: yesIndian Journal of Dermatology, 2008
Alopecia areata (AA) frequently occur in association with other autoimmune diseases such as thyroid disorders, anemias and other skin disorders with autoimmune etiology.
Thomas Emy, Kadyan R
doaj   +1 more source

Home - About - Disclaimer - Privacy