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An eruption associated with alphavirus infection
British Journal of Dermatology, 1996Some alphaviruses, e.g. Sindbis, cause an acute febrile illness associated with papular rashes and arthralgia. The diagnosis is usually serological and, hence, the histopathology of the rashes has been poorly elucidated. We report on two patients with rapidly healing eruptions associated with Sindbis virus infection.
K.M. Niemi, A.-L. Kariniemi, P. Autio
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Host responses to alphavirus infection
Immunological Reviews, 2008Summary: Members of the Alphavirus genus are classified into New World and Old World groups based upon their disease characteristics and primary areas of endemicity. The two groups exhibit noteworthy differences in pathogenesis during human infection, with Old World viruses primarily causing febrile and arthritogenic diseases and the New World viruses
William B. Klimstra, Kate D. Ryman
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The effects of alphavirus infection on neurons
Annals of Neurology, 1994Sindbis virus is an alphavirus that causes encephalitis in mice. The primary target cells for central nervous system infection are neurons. The outcome of neuronal infection is dependent on the age of the mouse at the time of infection (maturity of the neuron) and the strain of virus used for infection (virulence of the virus).
J. Marie Hardwick +3 more
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Animal models of alphavirus infection and human disease
2022Alphaviruses are a large group (>30 species) of enveloped, positive-strand RNA viruses. The re-emergence of mosquito-transmitted alphaviruses associated with human diseases ranging from severe and potentially fatal neurological disease to chronic arthritic disease highlights the need to understand the biology and pathogenesis of alphaviruses.
Cormac J, Lucas, Thomas E, Morrison
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Cross‐reactive Cytotoxic T Cells to Alphavirus Infection
Scandinavian Journal of Immunology, 1979Secondary Tc cells immune to alphaviruses (BEB, SIN and SFV) cross‐react between serologically defined subgroups at the level of target lysis and at the level of induction of response. Despite this apparently complete Tc cell cross‐reactivity between BFB, SIN and SFV, amisera raised against BEB and SIN showed virus specificity in their ability to block
Robert V. Blanden +2 more
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REGULATORS OF APOPTOSIS ON THE ROAD TO PERSISTENT ALPHAVIRUS INFECTION
Annual Review of Microbiology, 1997▪ Abstract Alphavirus infection can trigger the host cell to activate its genetically programmed cell death pathway, leading to the morphological features of apoptosis. The ability to activate this death pathway is dependent on both viral and cellular determinants. The more virulent strains of alphavirus induce apoptosis with increased efficiency both
Hardwick Jm, Griffin De
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Antibody-Mediated Clearance of Alphavirus Infection From Neurons
Science, 1991Humoral immunity is important for protection against viral infection and neutralization of extracellular virus, but clearance of virus from infected tissues is thought to be mediated solely by cellular immunity. However, in a SCID mouse model of persistent alphavirus encephalomyelitis, adoptive transfer of hyperimmune serum resulted in clearance of ...
Diane E. Griffin +5 more
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Alphavirus Infection in Cultured Tissue Cells
1991Mosquitoes have been recognized since the early 1900s as vectors of a variety of pathogenic viruses that have plagued humans and domestic animals for centuries. Thorough understanding of the life cycles of these pathogens is essential for developing methods for their control.
Dennis Brown, Mary L. Miller
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Duration of Alphavirus Neutralizing Antibody in Naturally Infected Birds
The American Journal of Tropical Medicine and Hygiene, 1988Native birds, mostly passerine species, ecologically associated with Culiseta melanura, the enzootic vector of eastern equine encephalomyelitis and Highlands J viruses in the eastern United States, were examined over a 12-year period in southeastern Massachusetts.
Kathleen S. Anderson +4 more
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Arthritis Associated with Alphavirus Infections: Chikungunya
2019Since 2004, the epidemic of alphavirus, mainly chikungunya viral (CHIKV) infection, has spread throughout the world and will continue to produce outbreaks due to viral mutations and the spread caused by travelers visiting endemic areas.
Jesús Sepúlveda-Delgado +6 more
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