Results 191 to 200 of about 37,722 (251)
Management of patients with protein s deficiency: focus on clinical course and direct oral anticoagulants. [PDF]
Altamura N +14 more
europepmc +1 more source
Congenital Protein C Deficiency Presenting as Neonatal Purpura Fulminans: A Report of Two Cases. [PDF]
Thaddanee R +4 more
europepmc +1 more source
Revisiting the Concept of DIC: A Phenotype-guided Framework for Modern Hemostatic Medicine. [PDF]
Crochemore T, Scarlatescu E.
europepmc +1 more source
Coagulation Markers and Myocardial Blush Grade in STEMI: Identifying Potential Predictors of Microvascular Reperfusion. [PDF]
Doğanözü E +6 more
europepmc +1 more source
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Thrombosis Research, 2018
Antithrombin is a key endogenous anticoagulant whose deficiency constitutes a strong risk factor for thrombosis. The study of antithrombin deficiency has generated excellent, and in some cases, surprising results that may be extrapolated to other thrombophilia and genetic disorders.
Javier Corral +2 more
exaly +3 more sources
Antithrombin is a key endogenous anticoagulant whose deficiency constitutes a strong risk factor for thrombosis. The study of antithrombin deficiency has generated excellent, and in some cases, surprising results that may be extrapolated to other thrombophilia and genetic disorders.
Javier Corral +2 more
exaly +3 more sources
Role of antithrombin concentrate in treatment of hereditary antithrombin deficiency
Thrombosis and Haemostasis, 2009SummaryAntithrombin (AT) functions as a potent natural anticoagulant and serine protease inhibitor that inactivates many enzymes in the coagulation cascade. Antithrombin also possesses anti-inflammatory properties, many of which are mediated by its actions as an anticoagulant.
George M Rodgers
exaly +3 more sources
Thrombosis Research, 1982
Plasma from two different thrombophilic families with functional inherited antithrombin III deficiency, i.e., with low antithrombin III activity but normal immunoreactive antithrombin III concentration, were investigated simultaneously in the same laboratory.
G Sas, Gy Blasko, T Kremmer
exaly +3 more sources
Plasma from two different thrombophilic families with functional inherited antithrombin III deficiency, i.e., with low antithrombin III activity but normal immunoreactive antithrombin III concentration, were investigated simultaneously in the same laboratory.
G Sas, Gy Blasko, T Kremmer
exaly +3 more sources
Monitoring of heparins in antithrombin-deficient patients [PDF]
Introduction: Heparins exert their anticoagulant effect through activation of antithrombin. Whether antithrombin deficiency leads to clinically relevantly reduced anti-Xa activity of heparins is unknown.
René Mulder +2 more
exaly +2 more sources
Thrombosis Research, 1987
Antithrombin III (AT III) microheterogeneity was investigated in 12 cases of congenital AT III deficiency and 2 cases of congenital AT III abnormality by isoelectric focusing (IEF) and immunofixation. In congenital AT III deficiency, IEF and immunofixation revealed AT III as 8 bands which was indistinguishable from normal control in terms of the number
N, Sakuragawa +4 more
openaire +2 more sources
Antithrombin III (AT III) microheterogeneity was investigated in 12 cases of congenital AT III deficiency and 2 cases of congenital AT III abnormality by isoelectric focusing (IEF) and immunofixation. In congenital AT III deficiency, IEF and immunofixation revealed AT III as 8 bands which was indistinguishable from normal control in terms of the number
N, Sakuragawa +4 more
openaire +2 more sources

