Results 181 to 190 of about 23,200 (255)

ELABELA: a polypeptide with therapeutic potential in cardiovascular diseases. [PDF]

Front Cardiovasc Med
Yan P, Meng C, Yang S, Huang W, Wang T.
europepmc   +1 more source

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Apelin/APJ system in inflammation

International Immunopharmacology, 2022
Apelin, an endogenous ligand for the G protein-coupled receptor (APJ), is widely distributed within the central nervous system and diverse organs in human and animals.
Yanjie Yang, Shaoping Ji, X W
exaly   +4 more sources

Neuroprotective gain of Apelin/APJ system.

Neuropeptides, 2021
Apelin is an endogenous ligand of G protein-coupled receptor APJ. In recent years, many studies have shown that the apelin/APJ system has neuroprotective properties, such as anti-inflammatory, anti-oxidative stress, anti-apoptosis, and regulating ...
Jiaxiu Zhou, Niannian Shuai, Bo Wang, Xin Jin, X. Kuang, S. Tian   +5 more
semanticscholar   +3 more sources

Role of Apelin/APJ axis in cancer development and progression.

Advances in Medical Sciences, 2020
Apelin is an endogenous peptide, which is expressed in a vast board of organs such as the brain, placenta, heart, lungs, kidneys, pancreas, testis, prostate and adipose tissues.
Javad Masoumi, A. Jafarzadeh, H. Khorramdelazad, Morteza Abbasloui, J. Abdolalizadeh, N. Jamali   +5 more
semanticscholar   +2 more sources

Apelin/APJ system and cancer

Clinica Chimica Acta, 2016
Apelin is an endogenous ligand of the apelin receptor (APJ), a seven-transmembrane G protein-coupled receptor. Apelin/APJ system has a wide tissue distribution in the brain as well as in peripheral organs including heart, lung, vessels, and adipose tissue.
Yanjie Yang, Shuang-Yu Lv, Liang Zhang
exaly   +3 more sources

Elabela-APJ axis attenuates cerebral ischemia/reperfusion injury by inhibiting neuronal ferroptosis.

Free Radical Biology & Medicine, 2023
Ferroptosis is a form of non-apoptotic cell death caused by iron-dependent peroxidation of lipids. It contributes to ischemic stroke-induced neuronal damage.
Pengfei Xu, Lingqi Kong, C. Tao, Yu-You Zhu, Juan Cheng, Wenyu Li, Nan Shen, Rui Li, Chao Zhang, Li Wang, Yan Zhang, Guo-Ping Wang, Xin-Feng Liu, Wen Sun, Wei Hu   +14 more
semanticscholar   +1 more source

APJ+ cells in the SHF contribute to the cells of aorta and pulmonary trunk through APJ signaling

Developmental Biology, 2023
Outflow tract (OFT) develops from cardiac progenitor cells in the second heart field (SHF) domain. APJ, a G-Protein Coupled Receptor, is expressed by cardiac progenitors in the SHF. By lineage tracing APJ+SHF cells, we show that these cardiac progenitors contribute to the cells of OFT, which eventually give rise to aorta and pulmonary trunk/artery upon
Kamal Baral, Gaetano D’amato, Bryce Kuschel, Frank Bogan, Brendan W. Jones, Colton L. Large, Jeffery D. Whatley, Kristy Red-Horse, Bikram Sharma   +8 more
openaire   +2 more sources