Results 191 to 200 of about 698,293 (229)
Some of the next articles are maybe not open access.
Apolipoprotein C-II Deficiency
1986The lipolysis of chylomicrons and very low density lipoproteins is responsible for the delivery of energy in the form of fatty acids to adipose or muscle tissue. The fatty acids are transported in the form of triacylglycerols in these lipoproteins following their synthesis in the intestine or liver.
A. L. Catapano, A. Capurso
openaire +3 more sources
Apolipoprotein C-II deficiency associated with nonfunctional mutant forms of apolipoprotein C-II
Canadian Journal of Biochemistry and Cell Biology, 1984Two previously unidentified apolipoproteins (apo) designated apo C-II-X and C-II-Y have been found in plasma of homozygotes and obligate heterozygotes of a family with apo C-II deficiency. Because the plasmas of homozygotes do not activate lipoprotein lipase, apo C-II-X and C-II-Y are apparently nonfunctional.
G F, Maguire +3 more
openaire +2 more sources
A purification method for apolipoprotein A-I and A-II
Analytical Biochemistry, 1989Apolipoproteins A-I and A-II were isolated from precipitates obtained by cold ethanol fractionation of human plasma. The starting material used in this report was precipitate B of the Kistler and Nitschmann method which corresponds approximately to fraction III of the Cohn and Oncley procedure.
Peitsch MC +5 more
openaire +3 more sources
Renal amyloidosis caused by a novel stop-codon mutation in the apolipoprotein A-II gene.
Kidney International, 2001BACKGROUND Although apolipoprotein A-II (apoA-II) associated amyloidosis has been described in the senescent accelerated mouse (SAM) model of aging, so far there has been no report of human apoA-II amyloidosis except for a recent report of renal ...
M. Yazaki +11 more
semanticscholar +1 more source
Apolipoprotein A-II modulates HDL remodeling in plasma
Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1992Native and reduced-carboxamidomethylated (RCM) HDL3 are incubated with a lipoprotein-depleted plasma fraction in the presence of triacylglycerol-rich particles isolated from Intralipid. Unmodified HDL3 are mainly converted into large HDL2b particles (diameter: 9.84 +/- 0.15 nm); RCM-HDL3 are transformed into both large HDL2b (9.76 +/- 0.10 nm) and ...
L. CALABRESI +3 more
openaire +2 more sources
Metabolism of apolipoproteins A-I, A-II, and A-IV
1986Publisher Summary Kinetic studies utilizing radioiodinated apolipoproteins have yielded important insights into lipoprotein metabolism. A major concern in these studies is that the tracer has identical kinetic behavior in vivo to native lipoproteins.
E J, Schaefer, J M, Ordovas
openaire +2 more sources
Apolipoprotein C-II Deficiency Revisited
New England Journal of Medicine, 1984In 1978, Breckenridge et al. described a patient with a deficiency of apolipoprotein C-II, the activator of the enzyme lipoprotein lipase.1 It was predicted that the newly discovered disorder would...
openaire +2 more sources
Kinetics of apolipoproteins A-I and A-II
1984Plasma lipoproteins are a polydisperse collection of particles which range in diameter from 7 to 160 nm. These lipoproteins particles have been classified according to methods of separation1. One system, based on density, divides the lipoproteins into five classes: chylomicrons (ρ < 0.95), very-low-density lipoproteins (VLDL, 0.95 < ρ < 1.006 ...
L. Zech +4 more
openaire +1 more source
Journal of Molecular Medicine, 1995
Screening for apolipoprotein (apo) C-II variants in the plasma of 400 students, 600 patients of a cardiological rehabilitation center, and 1200 patients of an outpatient lipid clinic by isoelectric focusing and subsequent anti-apo C-II immunoblotting led to the identification of four individuals whose plasma samples contained an apo C-II isoform with ...
H, Wiebusch +7 more
openaire +2 more sources
Screening for apolipoprotein (apo) C-II variants in the plasma of 400 students, 600 patients of a cardiological rehabilitation center, and 1200 patients of an outpatient lipid clinic by isoelectric focusing and subsequent anti-apo C-II immunoblotting led to the identification of four individuals whose plasma samples contained an apo C-II isoform with ...
H, Wiebusch +7 more
openaire +2 more sources
Metabolism, 1981
Familial apolipoprotein A-I and A-II deficiency (Tangier disease) is characterized by cholesterol ester deposition in histiocytes, decreased plasma cholesterol and low density lipoprotein cholesterol (C-LDL), and a striking deficiency of high density lipoproteins (HDL).
P, Alaupovic +4 more
openaire +2 more sources
Familial apolipoprotein A-I and A-II deficiency (Tangier disease) is characterized by cholesterol ester deposition in histiocytes, decreased plasma cholesterol and low density lipoprotein cholesterol (C-LDL), and a striking deficiency of high density lipoproteins (HDL).
P, Alaupovic +4 more
openaire +2 more sources

