Results 31 to 40 of about 73,331 (301)

Loss of human serum apolipoproteins C and E during manipulation of diluted solutions.

open access: yesJournal of Lipid Research, 1982
The adsorption of human serum very low density apolipoproteins C-I, C-II, C-III, and E to the interior surfaces or Beckman Airfuge plastic tubes and glass pipettes has been studied. 125I-Labeled apolipoprotein C-III-1 (1 microgram/ml) was used as a model
L Holmquist
doaj   +1 more source

Quantification of apolipoprotein E receptors in human brain-derived cell lines by real-time polymerase chain reaction

open access: yes, 2004
Apolipoprotein (apo) E4 is a risk factor for Alzheimer's disease (AD) and other neurodegenerative diseases, compared to wild-type apoE3. The mechanism(s) is unknown.
Owen, JS   +7 more
core   +1 more source

Competitive Enzyme Immunoassay for Apolipoprotein A-II [PDF]

open access: yes, 1982
Summary: A competitive enzyme immunoassay for apolipoprotein A-II was developed. Microtitre plates were used äs a solid phase and coated with anti-apolipoprotein A-II antibodies. Purified apolipoprotein A-II, labelled with horseradish peroxidase was used
K. Ilsemann   +5 more
core   +1 more source

Isolation and characterization of an apoA-II-containing lipoprotein (LP-A-II:B complex) from plasma very low density lipoproteins of patients with Tangier disease and type V hyperlipoproteinemia.

open access: yesJournal of Lipid Research, 1991
Previous studies have shown that very low density lipoproteins (VLDL) from patients with Tangier disease are less effective as a substrate for human milk lipoprotein lipase (LPL) than VLDL from normal controls as assessed by measuring the first order ...
P Alaupovic   +6 more
doaj   +1 more source

Distinct patterns of apolipoprotein C-I, C-II, and C-III isoforms are associated with markers of Alzheimer’s disease

open access: yesJournal of Lipid Research, 2021
Apolipoproteins C-I, C-II, and C-III interact with ApoE to regulate lipoprotein metabolism and contribute to Alzheimer's disease pathophysiology. In plasma, apoC-I and C-II exist as truncated isoforms, while apoC-III exhibits multiple glycoforms.
Yueming Hu   +4 more
doaj   +1 more source

Incorporation of a stable isotopically labeled amino acid into multiple human apolipoproteins.

open access: yesJournal of Lipid Research, 1991
Procedures are presented for the separation and determination of the isotopic enrichment of multiple human apolipoproteins labeled in vivo with a stable isotope amino acid.
BW Patterson   +4 more
doaj   +1 more source

Phospholipid complexation and association with apolipoprotein C-II: insights from mass spectrometry.

open access: yes, 2003
The interactions between phospholipid molecules in suspensions have been studied by using mass spectrometry. Electrospray mass spectra of homogeneous preparations formed from three different phospholipid molecules demonstrate that under certain ...
Ilag, LL   +5 more
core   +2 more sources

Apoc2 loss-of-function zebrafish mutant as a genetic model of hyperlipidemia

open access: yesDisease Models & Mechanisms, 2015
Apolipoprotein C-II (APOC2) is an obligatory activator of lipoprotein lipase. Human patients with APOC2 deficiency display severe hypertriglyceridemia while consuming a normal diet, often manifesting xanthomas, lipemia retinalis and pancreatitis ...
Chao Liu   +13 more
doaj   +1 more source

Integrating Proteomics and Lipidomics for Evaluating the Risk of Breast Cancer Progression: A Pilot Study

open access: yesBiomedicines, 2023
Metastasis is a serious and often life-threatening condition, representing the leading cause of death among women with breast cancer (BC). Although the current clinical classification of BC is well-established, the addition of minimally invasive ...
Natalia L. Starodubtseva   +13 more
doaj   +1 more source

Arginine-directed glycation and decreased HDL plasma concentration and functionality [PDF]

open access: yes, 2014
Background/Objectives: Decreased plasma concentration of high-density lipoprotein cholesterol (HDL-C) is a risk factor linked to increased risk of cardiovascular disease (CVD). Decreased anti-atherogenic properties of HDL are also implicated in increased
Rabbani, Naila   +4 more
core   +1 more source

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