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ATF6 protects against protein misfolding during cardiac hypertrophy
Journal of Molecular and Cellular CardiologyCardiomyocytes activate the unfolded protein response (UPR) transcription factor ATF6 during pressure overload-induced hypertrophic growth. The UPR is thought to increase ER protein folding capacity and maintain proteostasis. ATF6 deficiency during pressure overload leads to heart failure, suggesting that ATF6 protects against myocardial dysfunction by
Christoph, Hofmann +14 more
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ATF6 activation in breast cancer.
We studied mobilization of nuclear factors in human breast cancer.openaire +1 more source
The Unfolding Story of ATF6, Microbial Dysbiosis, and Colorectal Cancer
Gastroenterology, 2018Sydney, Lavoie, Wendy S, Garrett
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