Results 151 to 160 of about 5,680 (207)
Rgg/SHP transcriptional regulators, RaS-RiPPs, and their impacts in streptococci. [PDF]
Dey S, A Krivograd S, E Rued B.
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Ligand binding drives proteolysis of the SmcR master transcription factor and controls quorum sensing-state transitions in <i>Vibrio</i> species. [PDF]
Rasal TA +10 more
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Phages as Metabolic Switches in Plant-Associated Microbiomes: Implications for Climate-Smart Agriculture. [PDF]
Ali B +8 more
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Bacteria can communicate through an intercellular signaling system referred to as quorum sensing (QS). The QS system involves the production of autoinducers that interact with their respective receptors, leading to the induction of specific signal ...
Ronit Vogt Sionov +2 more
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The role of microbial dormancy autoinducers in metabolism blockade [PDF]
Alkyl-substituted hydroxybenzenes (AHBs), which are autoinducers of microbial dormancy (d1 factors), were found to stabilize the structure of protein macromolecules and modify the catalytic activity of enzymes.
Alexey Kolpakov +2 more
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Stabilization of enzymes by dormancy autoinducers as a possible mechanism of resistance of resting microbial forms [PDF]
Alkyl-substituted hydroxybenzenes (AHBs), autoinducers of microbial dormancy (or d1 factors), were found to stabilize the structure of protein macromolecules, making them metabolically less active and more resistant to stresses. In vitro experiments with
Alexey Kolpakov +2 more
exaly +1 more source
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Custom Synthesis of Autoinducers and Their Analogues
2010Bacterial quorum sensing (QS) system is a unique target for the development of a new class of drugs that potentially control pathogenicity and attenuate virulence. Thus, it has been of significant interest to discover small organic molecules that modulate QS circuits by competing with the signaling molecules, or so-called autoinducers (AIs), for ...
Jun, Igarashi, Hiroaki, Suga
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Autoinduced synthesis of colicin E2
Molecular and General Genetics MGG, 1983Escherichia coli K-12 cells carrying the high copy number plasmid ColE2-P9 and a sfiA-lacZ gene fusion exhibit abnormally high levels of SOS-regulated phi sfiA-lacZ expression. Increased sfiA-lacZ expression is caused by the action of colicin E2, which is a DNase, rather than by the presence of multiple copies of a binding site for LexA protein, the ...
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