Repair of Dihydrouracil Supported by Base Excision Repair in mNTH1 Knock-out Cell Extracts [PDF]
Rhoderick H. Elder, Grigory L. Dianov
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TOMM20 increases cancer aggressiveness by maintaining a reduced state with increased NADH and NADPH levels, oxidative phosphorylation (OXPHOS), and apoptosis resistance while reducing reactive oxygen species (ROS) levels. Conversely, CRISPR‐Cas9 knockdown of TOMM20 alters these cancer‐aggressive traits.
Ranakul Islam+9 more
wiley +1 more source
HMCES corrupts replication fork stability during base excision repair in homologous recombination-deficient cells. [PDF]
Peña-Gómez MJ+9 more
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Base excision repair is efficient in cells lacking poly(ADP-ribose) polymerase 1
Momchil D. Vodenicharov
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Induction of DNA polymerase beta-dependent base excision repair in response to oxidative stress in vivo [PDF]
Diane C. Cabelof
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Presurgery 72‐h fasting in GB patients leads to adaptations of plasma lipids and polar metabolites. Fasting reduces lysophosphatidylcholines and increases free fatty acids, shifts triglycerides toward long‐chain TGs and increases branched‐chain amino acids, alpha aminobutyric acid, and uric acid.
Iris Divé+7 more
wiley +1 more source
Beyond Nucleotide Excision Repair: The Importance of XPF in Base Excision Repair and Its Impact on Cancer, Inflammation, and Aging. [PDF]
Gohil D, Roy R.
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The role of aging and brain-derived neurotrophic factor signaling in expression of base excision repair genes in the human brain. [PDF]
Lautrup S+12 more
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Reconstitution of the base excision repair pathway for 7,8-dihydro-8-oxoguanine with purified human proteins [PDF]
Barbara Pascucci
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