Results 251 to 260 of about 73,773 (297)
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Beta Carotene in Congenital Porphyria
Archives of Dermatology, 1978To the Editor.— Dr George Stretcher, in his article on erythropoietic porphyria, which appeared in the NovemberArchives(113:1553-1557,1977), makes a reference to the lack of information about the long-term treatment with beta carotene. In 1973,I described a young woman, aged 18, with congenital porphyria at the Royal Society of Medicine ( Proc R Soc ...
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Lack of genotoxicity with beta-carotene
Toxicology Letters, 1988A literature review was conducted on adverse effects of carotenoids on human and animal development. The data suggest that beta-carotene administration prevented genetic damage caused by mutagens both in bacterial and cell culture systems, and that large doses of pure beta-carotene do not cause embryotoxicity in rodents.
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Beta-Carotene: Thermal Degradation
Science, 1964Thermal treatment of crystalline β-carotene (240°C in a vacuum) results in the formation of a volatile fraction containing chiefly aromatic hydrocarbons such as toluene, m - and p -xylene, 2,6-dimethylnaphthalene, and ionene.
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Effects of Dietary Protein, Fat and beta-Carotene Levels on beta-Carotene Absorption in Rats
International Journal for Vitamin and Nutrition Research, 2005It is important that the factors influencing functional carotenoid absorption be made clear. The effect of rat diets containing different combinations of dietary protein, fat, and beta-carotene levels on beta-carotene absorption was evaluated. In the mid-level beta-carotene diet groups (1.8 mg beta-carotene/100 g diet), the retinol content in the ...
Keisuke, Hosotani, Masahiro, Kitagawa
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Serum beta-carotene before and after beta-carotene supplementation.
European journal of clinical nutrition, 1992A two-month double-blind, placebo-controlled supplementation study of oral beta-carotene (20 mg daily) was conducted. Two hundred and twenty two 30-69 year old men were randomized into either a beta-carotene or placebo group, and serum samples were obtained at baseline, follow-up (2 months), and up to 12 weeks post-supplementation.
D, Albanes +6 more
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Beta-carotene accumulation in serum and skin
The American Journal of Clinical Nutrition, 1993The accumulation of beta-carotene in serum and skin was evaluated in human volunteers. A single 51-mg dose of beta-carotene given in the absence of dietary fat resulted in no detectable change in serum beta-carotene. The same dose administered with 200 g fat increased serum beta-carotene 2.5-fold at 40 h. Similarly, administering beta-carotene daily in
M R, Prince, J K, Frisoli
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Nutrition and cancer, 1989
Epidemiological studies have associated low dietary and/or plasma level of carotenoids with higher incidences of certain cancers. This evidence has led the National Cancer Institute to initiate more than a dozen prospective clinical trials in which supplements of beta-carotene alone, or in combination with other micronutrients, are being taken.
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Epidemiological studies have associated low dietary and/or plasma level of carotenoids with higher incidences of certain cancers. This evidence has led the National Cancer Institute to initiate more than a dozen prospective clinical trials in which supplements of beta-carotene alone, or in combination with other micronutrients, are being taken.
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1991
An infant is brought to the visiting American physician. She has seen children like this before and is not surprised when the interpreter tells her that the baby is sick and doesn’t open his eyes. A quick examination confirms her fears: the child is blind; behind his closed lids the corneas have dissolved into a shapeless jelly.
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An infant is brought to the visiting American physician. She has seen children like this before and is not surprised when the interpreter tells her that the baby is sick and doesn’t open his eyes. A quick examination confirms her fears: the child is blind; behind his closed lids the corneas have dissolved into a shapeless jelly.
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Safety of antioxidant vitamins and beta-carotene
The American Journal of Clinical Nutrition, 1995Epidemiologic evidence links high antioxidant status with low risk of degenerative disease. Optimal intakes of antioxidants may not be achievable by diet alone; supplements may be taken, particularly in subgroups of the population at high risk. It is thus necessary to ensure that antioxidant supplements are safe and free from side effects. The toxicity
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