Results 211 to 220 of about 109,357 (257)
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Pediatrics, 1954
Peripheral blood specimens from cases of varying degree of clinical severity of hemolytic disease of the newborn infant have been examined spectrophotometrically and a curve consistent with the presence of bilirubin was found in each case. Extracts of the pigmented brain areas from five cases of hemolytic disease of the newborn infant ...
W J, WATERS, D A, RICHERT, H H, RAWSON
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Peripheral blood specimens from cases of varying degree of clinical severity of hemolytic disease of the newborn infant have been examined spectrophotometrically and a curve consistent with the presence of bilirubin was found in each case. Extracts of the pigmented brain areas from five cases of hemolytic disease of the newborn infant ...
W J, WATERS, D A, RICHERT, H H, RAWSON
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Neuropathology and Applied Neurobiology, 1983
In babies of low birth weight dying in the first week of life, bilirubin encephalopathy involving the thalamus only or the thalamus and one or two other areas occurred in nineteen of 376 cases examined over a 10 year period. Although coexistent germinal layer haemorrhage was present in sixteen, this was not thought to be an aetiological factor.
A J, Sherwood, J F, Smith
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In babies of low birth weight dying in the first week of life, bilirubin encephalopathy involving the thalamus only or the thalamus and one or two other areas occurred in nineteen of 376 cases examined over a 10 year period. Although coexistent germinal layer haemorrhage was present in sixteen, this was not thought to be an aetiological factor.
A J, Sherwood, J F, Smith
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Prevention of Bilirubin Encephalopathy
Neonatology, 2001Prevention of bilirubin encephalopathy is based on the detection of infants at risk of developing a significant hyperbilirubinemia. This task can be accomplished by performing a simple umbilical cord blood test, such as blood group, Rh, Coombs’ test and glucose-6-phosphate dehydrogenase, in order to detect hemolytic diseases.
BERTINI G. +3 more
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Pediatrics, 1955
(1) Staining of the brain can be produced in normal newborn white rats by intraperitoneal injection of a solution of bilirubin. (2) Under similar conditions it was not possible to produce staining of the brain in a comparable series of normal adult rats. (3) This study further emphasizes the importance of the immaturity of
William J. Waters, Howard A. Britton
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(1) Staining of the brain can be produced in normal newborn white rats by intraperitoneal injection of a solution of bilirubin. (2) Under similar conditions it was not possible to produce staining of the brain in a comparable series of normal adult rats. (3) This study further emphasizes the importance of the immaturity of
William J. Waters, Howard A. Britton
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Chemoprevention of bilirubin encephalopathy with a nanoceutical agent
Pediatric Research, 2022Targeted rapid degradation of bilirubin has the potential to thwart incipient bilirubin encephalopathy. We investigated a novel spinel-structured citrate-functionalized trimanganese tetroxide nanoparticle (C-Mn3O4 NP, the nanodrug) to degrade both systemic and neural bilirubin loads.Severe neonatal unconjugated hyperbilirubinemia (SNH) was induced in ...
Aniruddha, Adhikari +11 more
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Quantitative ADC in bilirubin encephalopathy
Japanese Journal of Radiology, 2013It was with great interest that we read the paper by Cece and colleagues [1] entitled ‘Diffusion-weighted imaging of patients with neonatal bilirubin encephalopathy.’ The authors conclude that quantitative apparent diffusion coefficient (ADC) measurement could be a promising marker of neonatal bilirubin encephalopathy.
Jon F, Watchko +2 more
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The prevention of bilirubin encephalopathy
The Journal of Pediatrics, 1958Summary 1. Maternal titer values and/or apositive Coombs' test by themselves are not an indication for replacement transfusion in hemolytic disease of the newborn due to Rh incompatibility. The main emphasis should be placed on the clinical condition and early course of the newborn infant, particularly the ability to handle bilirubin. 2.
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Clinical Features of Bilirubin Encephalopathy
Clinics in Perinatology, 1990Clinical features of bilirubin encephalopathy vary depending on the age of the infant and the degree of hyperbilirubinemia. In term infants with hyperbilirubinemia, three distinct clinical phases are apparent in the first weeks of life, and long-term consequences include extrapyramidal disturbances (particularly athetosis), hearing loss, gaze ...
A M, Connolly, J J, Volpe
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Bilirubin encephalopathy: Changing concepts
Brain and Development, 1980This overview begins with a discussion of clinical approaches to prevent brain damage in jaundiced newborns. Results of longitudinal follow-up studies are given. The main part deals with pathogenesis, whereby bilirubin albumin binding, competition by drugs, bilirubin distribution and equilibration in the body, the increased sensitivity of the infantile
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Acute, Severe Bilirubin Encephalopathy in a Newborn
Pediatric Emergency Care, 2004In recent years, changes in health care practices including the early discharge of newborns have transformed the management of neonatal jaundice into an outpatient problem. At the same time, there has been a resurgence in the incidence of kernicterus.
Thomas J, Mollen +2 more
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