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Oxidative Phosphorylation in Experimental Bilirubin Encephalopathy
Science, 1967Mitochondria from the whole brain or cerebellum of newborn guinea pigs with experimental bilirubin encephalopathy failed to exhibit uncoupling of oxidative phosphorylation. The pigment concentrations required to initiate uncoupling in vitro are much higher than those found in the brain of neurotoxic animals.
I, Diamond, R, Schmid
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Kernicterus (Bilirubin Encephalopathy)
1975The first report of kernicterus is generally attributed to Orth (1875), although his description is somewhat unclear and emphasizes staining of the superficial portions of brain tissue in the ventricular walls and at the hemispheric surface. The first comprehensive description of the disease was given by Schmorl (1903) who distinguished two types of ...
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Neonatal Jaundice and Prevention of Bilirubin Encephalopathy
Postgraduate Medicine, 1959The physician should be alert to the development of jaundice in the newborn infant. A high concentration of serum bilirubin may cause injury to the brain. Blood group incompatibility, infection, hepatitis or congenital anomalies of the biliary tree may result in the development of jaundice. Other less well-known factors also may be responsible.
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International Journal of Pharmacognosy and Pharmaceutical Sciences, 2021
R Vinola +5 more
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R Vinola +5 more
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Bilirubin Encephalopathy (Kernicterus)
2015AbstractThe striking correlation between jaundice, elevated serum bilirubin levels and brain damage with pigmentation has been well documented by several investigators in relation to hemolytic ...
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Zhurnal nevropatologii i psikhiatrii imeni S.S. Korsakova (Moscow, Russia : 1952), 1990
The author describes symptoms typical for children with bilirubin encephalopathy (the hyperkinetic patterns of infantile cerebral paralysis, audition deficiency, mental deficiency). During follow up studies (from 1 month or 1-5 years and up to 10-25 years) of children with bilirubin encephalopathy uncomplicated by prolonged asphyxia and derangement of ...
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The author describes symptoms typical for children with bilirubin encephalopathy (the hyperkinetic patterns of infantile cerebral paralysis, audition deficiency, mental deficiency). During follow up studies (from 1 month or 1-5 years and up to 10-25 years) of children with bilirubin encephalopathy uncomplicated by prolonged asphyxia and derangement of ...
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Mitochondrial Abnormalities in Bilirubin Encephalopathy
Journal of Neuropathology and Experimental Neurology, 1970H S, Schutta, L, Johnson, H E, Neville
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Neuroglobin expression and function in the temporal cortex of bilirubin encephalopathy rats
Anatomical Record, 2022Jin Xu, Gan Sheng-Wei, Juan Huang
exaly

