Results 131 to 140 of about 2,964 (168)
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Borna disease virus and deficit schizophrenia
Acta Neuropsychiatrica, 2003Background:It is controversial whether Borna disease virus (BDV) infects humans and causes psychiatric disorders.Objectives:The relationship between BDV infection and schizophrenia with deficit syndrome was investigated.Study design:Using the Schedule for the Deficit Syndrome, 62 schizophrenic in-patients were selected from three psychiatric hospitals.
Yong-Ku, Kim +9 more
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Molecular Biology of Borna Disease Virus
1995Originally described in the early nineteenth century as a fatal encephalitis in horses, Borna disease (BD) has become an extraordinarily valuable model for the study of both molecular mechanisms and biological consequences of persistent virus infection in the CNS (Nicolau and Galloway 1928; Zwick 1939, this volume).
T, Briese, W I, Lipkin, J C, de la Torre
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Genetic Relationship of Borna Disease Virus Isolates
Virus Genes, 2003The infection of humans with Boma disease virus (BDV) is still a matter of debate. In a recent publication, we described a BDV (RW98) isolated from the blood of a psychiatric patient. The RNA of this virus differed more than 5% from that of the widely used strain He/80, which was supposed to represent our laboratory virus.
Oliver, Planz +2 more
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Borna disease virus interference with neuronal plasticity
Virus Research, 2005Viruses able to infect the central nervous system (CNS) are increasingly being recognized as important factors that can cause mental diseases by interfering with neuronal plasticity. The mechanisms whereby such infections disturb brain functions are beginning to emerge.
Gonzalez-Dunia, Daniel +3 more
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2016
The neurotropic virus, Borna disease virus (BDV), a member of a group of nonsegmented, negative strand ribonucleic acid (RNA) viruses (order Mononegavirales), infects warm-blooded animal species. Infection among mammals may be asymptomatic, produce neurobehavioral abnormalities, or result in fatal meningoencephalitis.
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The neurotropic virus, Borna disease virus (BDV), a member of a group of nonsegmented, negative strand ribonucleic acid (RNA) viruses (order Mononegavirales), infects warm-blooded animal species. Infection among mammals may be asymptomatic, produce neurobehavioral abnormalities, or result in fatal meningoencephalitis.
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Sequence characterization of human Borna disease virus
Virus Research, 1996Borna disease virus (BDV) causes a central nervous system disease in several vertebrate animal species, which is manifest by behavioral abnormalities. Seroepidemiologic data suggest that BDV might infect humans, possibly being associated with certain mental disorders.
J C, de la Torre +4 more
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Replication of Borna disease virus in cell cultures
Medical Microbiology and Immunology, 1980Borna disease (BD) virus from infected brain tissue of horses or rabbits could be grown in embryonic brain cells from rabbits or rats with high virus yields. The cells became persistently infected and could be subcultivated without loss of infectivity.
S, Herzog, R, Rott
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Activated Borna Disease Virus in Affective Disorders
Pharmacopsychiatry, 1999Borna disease virus (BDV) is an animal pathogen that causes behavioral changes in animals. Previous studies have found a high prevalence of serum antibodies as well as Borna disease viral antigens (BDVAGs) and RNA in the white blood cells of psychiatric patients, especially those with affective disorders.
R, Ferszt +6 more
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Borna Disease Virus infection in young children
APMIS, 2008Borna Disease Virus infection (BDV) is a well known animal disease. It got its name from a disease outbreak among horses around the small town ‘‘Borna’’, 26 km south-east of Leipzig in the late 19th century. The symptoms in horses have been described in detail and involve neurologic as well as mental and behavioral disorders.
Thomas, Scholbach, Liv, Bode
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Borna disease virus and depression
Trends in Microbiology, 2001High amounts of circulating immune complexes of Borna disease virus correlates with severity of depression, according to Dr Liv Bode of the Robert Koch-Institut in Berlin, Germany. The virus targets limbic structure neurons and is known to cause behavioral abnormalities in animals.
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