Results 291 to 300 of about 68,566 (325)

Hypertension in Living Kidney Donors Has No Effect on Complement Activation and Fibrosis.

Kidney Blood Press Res
Wagner N, Angeloni M, Ferrazzi F, Müller-Deile J, Büttner-Herold M, Amann K, Daniel C, Vonbrunn E.   +7 more
europepmc   +1 more source

Complement biosynthesis in human brain: Insights from single-nucleus transcriptomics of hippocampus

Nguyen VD, Bright M, Zhou Y, Morgan BP, Zelek WM.   +4 more
europepmc   +1 more source

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Paeoniflorin prevents aberrant proliferation and differentiation of intestinal stem cells by controlling C1q release from macrophages in chronic colitis.

Pharmacological Research, 2022
The pathological features of inflammatory bowel disease necessitate therapeutic strategies aimed at restoring intestinal mucosal barrier function in addition to controlling inflammation. Paeoniflorin, a bioactive herbal constituent isolated from the root
Yirui Wang, Keyuan You, Yan You, Qian Li, Gui-ze Feng, Jiahui Ni, Xinyue Cao, Xiaowen Zhang, Yan-hang Wang, Weilian Bao, Xu Wang, Tongqing Chen, Haidong Li, Yuran Huang, Jiaren Lyu, Shi Yu, Hong Li, Suowen Xu, Kewu Zeng, Xiaoyan Shen   +19 more
semanticscholar   +1 more source

C1q, antibodies and anti-C1q autoantibodies

Molecular Immunology, 2015
The complement system has long been known for its role in combating infections. More recently the complement system is becoming increasingly appreciated for its role in processes that range from waste transport, immune tolerance and shaping of the adaptive immune response.
Beurskens, F.J., Schaarenburg, R.A. van, Trouw, L.A.   +2 more
openaire   +4 more sources

C1q-binding proteins and C1q receptors

Current Opinion in Immunology, 1999
Aggregated or immobilized complement C1q induces cellular responses in many different cell types. C1q-induced cellular responses may be involved in host defense and in protection against autoimmunity because C1q-deficient humans have infectious complications and a very high incidence of autoimmune disease.
Lloyd B. Klickstein, Anne Nicholson-Weller   +1 more
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Macrophage C1q: characterization of a membrane form of C1q and of multimers of C1q subunits.

The Journal of Immunology, 1987
Abstract It has been shown recently that C1q, a subcomponent of the first component of the classical complement pathway, is synthesized by macrophages and that endogenous C1q is detectable on the macrophage membrane. In this report, we demonstrate that membrane-associated C1q, which contains the A, B, and C chains of C1q ...
H, Martin, H P, Heinz, K, Reske, M, Loos
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C1q binding and C1q deviation assays using enzyme labelled C1q

Journal of Immunological Methods, 1984
Methods for the enzyme labelling of C1q are described. The C1q conjugates have been incorporated into suitably modified C1q deviation and C1q binding assays for circulating immune complexes. These assays show the expected high positivity rate in sera from patients with systemic lupus erythematosus.
G.W. Smith, I.J. Simpson, M.J. Myles
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RAGE binds C1q and enhances C1q-mediated phagocytosis

Cellular Immunology, 2012
RAGE, the multiligand receptor of the immunoglobulin superfamily of cell surface molecules, is implicated in innate and adaptive immunity. Complement component C1q serves roles in complement activation and antibody-independent opsonization. Using soluble forms of RAGE (sRAGE) and RAGE-expressing cells, we determined that RAGE is a native C1q globular ...
Fei Song, Vivek Rai, Barry I. Hudson, Ann Marie Schmidt, Wanchao Ma, Gaetano R. Barile   +5 more
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C1Q nephropathy in children

Pediatric Nephrology, 2005
C1q nephropathy (C1qNP) is a peculiar form of glomerulonephritis characterized by mesangial immunoglobulin and complement deposits, predominantly C1q, with no evidence of systemic lupus erythematosus. We describe the incidence, manifestation, histopathologic findings, follow-up, treatment and outcome of C1qNP.
Mojca Avguštin Čavić, Alenka Vizjak, Rajko B. Kenda, Tanja Kersnik Levart, A Hvala, Dušan Ferluga   +5 more
openaire   +3 more sources