Results 31 to 40 of about 9,499 (196)

Determination of Flunixin Meglumine in Veterinary Pharmaceutical Formulations by Capillary Electrophoresis With Capacitively Coupled Contactless Conductivity Detection. [PDF]

open access: yesElectrophoresis
A CE‐C4D method was developed for the indirect determination of flunixin meglumine (FM) by quantifying meglumine and using lithium ions as an internal standard. The method allows the use of a low‐cost standard reagent, providing suitable analytical performance for the determination of FM in veterinary pharmaceutical formulations.
de Araujo LM, da Silva JAF, de Jesus DP.
europepmc   +2 more sources

Arteriolar C4d in IgA Nephropathy: A Cohort Study [PDF]

open access: yesAmerican Journal of Kidney Diseases, 2020
Glomerular C4d (C4dG) as an indicator of the lectin pathway of complement activation in immunoglobulin A nephropathy (IgAN) has been associated with more severe kidney damage. Recent studies have suggested that vascular lesions in IgAN biopsy specimens with complement deposition are also associated with disease progression.
Faria, B.   +10 more
openaire   +3 more sources

Clinicopathological Associations of Plasma Cell-Rich Rejection in Pediatric Liver Transplant Recipients. [PDF]

open access: yesPediatr Transplant
Plasma cell‐rich rejection (PCRR) occurred in 15.3% of pediatric liver transplant recipients. PCRR was associated with recurrent rejection, severe rejection, central perivenulitis, ANA positivity, and chronic rejection. PCRR was not independently associated with mortality.
Gemell R   +3 more
europepmc   +2 more sources

CAQ Corner: Immune‐mediated complications

open access: yes, 2022
Liver Transplantation, EarlyView.
Mary Thomson, John R. Lake
wiley   +1 more source

Non-invasive monitoring of renal transplant recipients: Urinary excretion of soluble adhesion molecules and of the complement-split product C4d [PDF]

open access: yes, 2003
Background: The number of inducible adhesion molecules known to be involved in cell-mediated allograft rejection is still increasing. In addition, recent data describe complement activation during acute humoral allograft rejection.
Lederer, Stephan R.   +5 more
core   +1 more source

C4d immunohistochemistry in membranous nephropathy

open access: yesJournal of Laboratory Physicians, 2014
ABSTRACT Background: Membranous nephropathy (MN) is the most common cause of nephropathy in adults. The diagnosis is based on characteristic light microscopic, electron microscope and immunofluorescence (IF) findings. In early MN, the light microscopic findings may be difficult to differentiate from minimal chain disease.
Hui, Monalisa   +4 more
openaire   +2 more sources

Clinical and pathological correlations of C4d immunostaining and its infl uence on the outcome of kidney transplant recipients

open access: yesBrazilian Journal of Nephrology, 2011
INTRODUCTION: C4d is a marker of antibody-mediated rejection (ABMR) in kidney allografts, although cellular rejection also have C4d deposits. OBJECTIVE: To correlate C4d expression with clinico-pathological parameters and graft outcomes at three years ...
Virna Nowotny Carpio   +7 more
doaj   +1 more source

Role of complement activation product C4d as a predictor biomarker in lung cancer diagnosis: a case–control study

open access: yesEgyptian Journal of Chest Disease and Tuberculosis, 2021
Background Molecular biomarkers such as complement C4d in bronchoalveolar lavage (BAL) may interfere with lung cancer diagnosis. However, limited studies have been conducted.
Lucy A El-Maboud Suliman   +3 more
doaj   +1 more source

Renal Transplant Patients Biopsied for Cause and Tested for C4d, DSA, and IgG Subclasses and C1q: Which Humoral Markers Improve Diagnosis and Outcomes?

open access: yesJournal of Immunology Research, 2017
The association between donor specific antibodies (DSA) and renal transplant rejection has been generally established, but there are cases when a DSA is present without rejection.
James C. Cicciarelli   +9 more
doaj   +1 more source

C4d as a Diagnostic Tool in Proliferative GN [PDF]

open access: yesJournal of the American Society of Nephrology, 2015
Proliferative GN is classified as immune complex-mediated or complement-mediated (C3 glomerulopathy). Immune complex-mediated GN results from glomerular deposition of immune-complexes/Ig and C3; the C3 is derived from activation of the classical and/or lectin pathways of complement.
Sanjeev, Sethi   +3 more
openaire   +2 more sources

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