Results 211 to 220 of about 53,962 (242)
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Determination of Iron in Caco-2 cells by ET-AAS

Analytical and Bioanalytical Chemistry, 2005
An electrothermal atomic absorption spectrometric method (ET-AAS) was developed for the direct determination of iron in intestinal Caco-2 cells after studying cell viability and proliferation using 3-(4,5-dimethylthiazole-2-yl)-2,5-diphenyl tetrazolium bromide (MTT test).
Zeiner, Michaela   +4 more
openaire   +2 more sources

Campylobacter jejuni motility and invasion of Caco-2 cells

Infection and Immunity, 1995
We investigated the influence of motility on Campylobacter jejuni binding and invasion of Caco-2 cells. C. jejuni was motile in soft agar at basic (pH 8.5) and neutral pH values representative of the intestinal environment. However, C. jejuni was immobilized at pH 5.0. The inability of C.
C M, Szymanski   +3 more
openaire   +2 more sources

Toxic and biochemical effects of zinc in Caco-2 cells

Journal of Inorganic Biochemistry, 2003
Zinc (in relatively high concentrations) can be toxic to intestinal cells. The aim of the present study was to quanitfy cellular injury in preconfluent, colonic cancerous cells and in postconfluent, differentiating human intestinal Caco-2 cells. Cellular damage was measured by using cell proliferation, lactate dehydrogenase (LDH)-release, and apoptosis
Zödl, Bettina   +6 more
openaire   +3 more sources

Putrescine transport in differentiated Caco-2 cells.

Gastroenterologie clinique et biologique, 1996
To evaluate the putative importance of digestive luminal polyamines, we investigated the role of oxidation in putrescine uptake and transport in differentiated Caco-2 cells grown in monolayer.Our results confirm a putrescine uptake system located both at the apical and basolateral cell membrane. Putrescine is efficiently transported and excreted in the
Barlier, A.M.   +2 more
openaire   +2 more sources

Novel Choline Transport Characteristics in Caco-2 Cells

Drug Development and Industrial Pharmacy, 2002
Choline transport is characterized by sodium-dependent high-affinity, sodium-independent low-affinity, and sodium-independent blood-brain barrier transport mechanisms. Each defined mechanism has specific characteristics with regard to affinity for choline, transport capacity, and inhibition by hemicholinium.
A P, Crowe   +3 more
openaire   +2 more sources

Competition Effects on Carotenoid Absorption by Caco-2 Cells

2009
International ...
Reboul, Emmanuelle, Borel, Patrick
openaire   +3 more sources

Transepithelial Taurine Transport in Caco-2 Cell Monolayers

Journal of Membrane Biology, 2005
Here we characterized transepithelial taurine transport in monolayers of cultured human intestinal Caco-2 cells by analyzing kinetic apical and basolateral uptake and efflux parameters. Basolateral uptake was Na(+)- and Cl(-)- dependent and was inhibited by beta-amino acids.
S, Roig-Pérez, M, Moretó, R, Ferrer
openaire   +2 more sources

Poliovirus infection induces apoptosis in CaCo-2 cells

Journal of Medical Virology, 1999
The effects of poliovirus infection in CaCo-2 cells, a human enterocyte-like cell line are described. Infected cells were examined by a combination of techniques, including optical and electron microscopy, cytofluorimetric analysis of DNA content, and DNA agarose gel electrophoresis. Results obtained by the different experimental approaches demonstrate
M G, Ammendolia   +3 more
openaire   +2 more sources

Transport of curcumin derivatives in Caco-2 cell monolayers

European Journal of Pharmaceutics and Biopharmaceutics, 2017
Curcumin (Cur) is a strong natural antioxidant, who can prevent multiple diseases such as anti-cancer, anti-inflammatory, have a resistance to alzheimer's disease and various malignant diseases. But it has poor oral bioavailability due to its poor aqueous solubility, as well as instability.
Zhen, Zeng   +6 more
openaire   +2 more sources

Cholesterol Trafficking in CaCo-2 Cells

1998
In CaCo-2 cells, the transport of cholesterol from the plasma membrane to the endoplasmic reticulum occurs via transport vesicles, likely derived from specialized domains of the plasma membrane. Agents that inhibit p-glycoprotein activity interfere with the transport of plasma membrane cholesterol to the endoplasmic reticulum and the secretion of ...
openaire   +1 more source

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