Results 61 to 70 of about 28,628 (268)

Superficial siderosis in cerebral amyloid angiopathy

Annals of Indian Academy of Neurology, 2011
Wani Nisar, Kosar Tasleem, Rawa Aijaz, Qayum Abdul   +3 more
doaj   +1 more source

Identifying potential drug targets for cerebral amyloid angiopathy: A Mendelian randomization study based on the druggable genes. [PDF]

Medicine (Baltimore)
Zhang J, Xie A, Yan Z.
europepmc   +2 more sources

Interpretable classification of Alzheimer's disease pathologies with a convolutional neural network pipeline. [PDF]

, 2018
Neuropathologists assess vast brain areas to identify diverse and subtly-differentiated morphologies. Standard semi-quantitative scoring approaches, however, are coarse-grained and lack precise neuroanatomic localization.
Beckett, Laurel, Chuang, Kangway V, DeCarli, Charles, Dugger, Brittany N, Jin, Lee-Way, Keiser, Michael J, Tang, Ziqi   +6 more
core  

Coupling CZE, Liquid‐Phase Ion Mobility, to MS/MS for Quantitative Top‐Down Proteomics: Revealing Significant Proteoform Differences Between Healthy and Alzheimer's Disease Brains

PROTEOMICS, EarlyView.
ABSTRACT Alzheimer's disease (AD) is a neurodegenerative disorder characterized by cognitive decline and pathological protein aggregation. Comprehensive quantitative proteomics of brain tissues from AD patients is critical for pursuing a better understanding of the molecular mechanisms that drive AD progression.
Mehrdad Falamarzi Askarani, Fei Fang, Scott E. Counts, Liangliang Sun   +3 more
wiley   +1 more source

New Therapeutic Approaches for Alzheimer’s Disease and Cerebral Amyloid Angiopathy [PDF]

, 2014
Accumulating evidence has shown a strong relationship between Alzheimer’s disease (AD), cerebral amyloid angiopathy (CAA), and cerebrovascular disease.
Masafumi Ihara, Satoshi Saito
core   +2 more sources

Migrasomes, Matrix‐Bound Nanovesicles, and More: Messengers in the Matrix

PROTEOMICS, EarlyView.
ABSTRACT Extracellular vesicles (EVs) and particles (EPs) are diverse micro‐ and nanoparticles that circulate in bodily fluids and can attach to, or be deposited onto, the extracellular matrix (ECM) and other surfaces. To date, the nomenclature and classification of matrix‐bound or matrix‐associated EVs and EPs (MEVPs) have been unclear, largely due to
Anna V. Kolesov, Natalie Reitz, Marley Dewey, Colin L. Hisey   +3 more
wiley   +1 more source

Amyloid Spells and High Blood Pressure: Imminent Danger

Case Reports in Neurology, 2014
We present the case of a 71-year-old male, admitted after a generalized tonic-clonic seizure, with a history of recurrent left arm and face paresthesias, associated with sulcal cortical subarachnoid hemorrhages. During the next 48 h, he remained agitated
Andre Caetano, Miguel Pinto, Sofia Calado, Miguel Viana-Baptista   +3 more
doaj   +1 more source

Noninvasive imaging of amyloid‐beta and tau in rodent and nonhuman primate models

VIEW, EarlyView.
Imaging of amyloid‐beta plaque and tau accumulation in rodent and nonhuman primate model of Alzheimer's disease. Created in BioRender. Ni R. 2026. https://BioRender.com/a97h5ec Abstract Neurodegenerative diseases are characterized by the aberrant accumulation of protein aggregates.
Ruiqing Ni, Axel Rominger
wiley   +1 more source

Cerebral Amyloid Angiopathy

Türk Nöroloji Dergisi, 2009
Cerebral amyloid angiopathy (CAA) is characterized by the accumulation of amyloid beta-peptides (Ab) in the walls of leptomeningeal arteries, arterioles, and veins.
Mahmut Edip Gürol
doaj  

Cerebrospinal fluid biomarkers in cerebral amyloid angiopathy: insights from a clinical case series

Arquivos de Neuro-Psiquiatria
Cerebral amyloid angiopathy (CAA) is a small vessel disease characterized by the deposition of amyloid-beta in small cerebral vessels, which can lead to intracerebral hemorrhages and cognitive impairment.
Ana Silvia Sobreira Lima Verde, Alessandra Braga Cruz Guedes de Morais, Amanda Vale Catunda, João Igor Dantas Landim, Ian Silva Ribeiro, Bruno Diógenes Iepsen, Norberto Anízio Ferreira Frota   +6 more
doaj   +1 more source