Results 71 to 80 of about 26,516 (298)

Chylomicrons promote intestinal absorption of lipopolysaccharides

open access: yesJournal of Lipid Research, 2009
Recent data suggest that dietary fat promotes intestinal absorption of lipopolysaccharides (LPS) from the gut microflora, which might contribute to various inflammatory disorders.
Sarbani Ghoshal   +4 more
doaj  

Metabolic fate of fructose ingested with and without glucose in a mixed meal. [PDF]

open access: yes, 2014
Ingestion of pure fructose stimulates de novo lipogenesis and gluconeogenesis. This may however not be relevant to typical nutritional situations, where fructose is invariably ingested with glucose.
de Giorgi, S.   +7 more
core   +1 more source

Hepatic Lipidomics Unravels the Lipid‐Lowering and Anti‐Obesity Efficacy of Diacylglycerol Oil: Mechanistic Insights From High‐Fat Diet‐Induced Obese Mice

open access: yesFood Science &Nutrition, Volume 13, Issue 6, June 2025.
In a 12‐week clinical trial, DAG reduced serum TG and LDL cholesterol in obese individuals. In high‐fat diet mice, DAG lowered lipids, visceral fat, and hepatic steatosis, acting via inhibiting lipid synthesis (SCD1, FASN) and promoting fatty acid oxidation (CPT‐1) while modulating glycerophospholipid metabolism.
Lina Shi   +7 more
wiley   +1 more source

Differences in the processing of chylomicron remnants and beta-VLDL by macrophages.

open access: yesJournal of Lipid Research, 1990
To gain a detailed understanding of those factors that govern the processing of dietary-derived lipoprotein remnants by macrophages we examined the uptake and degradation of rat triacylglycerol-rich chylomicron remnants and rat cholesterol-rich beta-very
JL Ellsworth   +3 more
doaj  

Luminal Lipid Regulates CD36 Levels and Downstream Signaling to Stimulate Chylomicron Synthesis*

open access: yesJournal of Biological Chemistry, 2011
The membrane glycoprotein CD36 binds nanomolar concentrations of long chain fatty acids (LCFA) and is highly expressed on the luminal surface of enterocytes. CD36 deficiency reduces chylomicron production through unknown mechanisms.
T. Tran   +11 more
semanticscholar   +1 more source

Immunochemical studies on human plasma lipoproteins [PDF]

open access: yes, 1957
Some years ago, a series of human serum lipoproteins, distinguishable by their hydrated densities and lipid-protein ratios were recognized and isolated by ultracentrifugal techniques (1).
Aladjem, Frederick   +2 more
core   +1 more source

Vitamin K: Metabolism, Genetic Influences, and Chronic Disease Outcomes

open access: yesFood Science &Nutrition, Volume 13, Issue 6, June 2025.
Vitamin K exists in two natural isoforms: phylloquinone (PK, vitamin K1) and menaquinones (MKs, vitamin K2), and is an essential cofactor for the post‐translational carboxylation of vitamin K‐dependent proteins (VKDPs). VKDPs are implicated in various physiological processes, including blood coagulation, calcium homeostasis, as well as metabolic and ...
Montana Dupuy   +7 more
wiley   +1 more source

Hepatic lipase treatment of chylomicron remnants increases exposure of apolipoprotein E.

open access: yesJournal of Lipid Research, 1993
The consequences of hepatic lipase treatment of chylomicron remnants were studied. Rats were fed corn oil to induce production and secretion of chylomicrons and were then injected with polyclonal antiserum raised against hepatic lipase to specifically ...
DL Brasaemle   +2 more
doaj  

Endotoxin increase after fat overload is related to postprandial hypertriglyceridemia in morbidly obese patients. [PDF]

open access: yes, 2017
The low-grade inflammation observed in obesity has been associated with a high-fat diet, though this relation is not fully understood. Bacterial endotoxin, produced by gut microbiota, may be the linking factor.
Andrés Lacueva, Ma. Cristina   +7 more
core   +1 more source

Genetically Predicted Gut Microbiota and Host Metabolites Mediate the Causal Link Between Dietary Factors and Acute Myeloid Leukemia

open access: yesFood Science &Nutrition, Volume 13, Issue 6, June 2025.
Gut microbial and metabolite pathways mediate the dietary influence on AML risk. Processed meat intake was associated with increased AML via L‐histidine degradation. Conversely, higher glycerol and a greater free cholesterol‐to‐total lipid ratio in large HDL correlated with reduced AML risk.
Jiading Qin   +4 more
wiley   +1 more source

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