Results 231 to 240 of about 58,125 (347)

Rapid and Accurate Genotoxicity Assessment Using the Neutral Comet Assay in <i>Cyprinus carpio</i> Cells. [PDF]

open access: yesLife (Basel)
So B   +11 more
europepmc   +1 more source

Contemporary disasters may not kill more women than men: an empirical inquiry into sex‐differentiated fatalities in the twenty‐first century

open access: yesDisasters, Volume 50, Issue 3, July 2026.
Abstract This study investigates the claim that women are disproportionately more likely to die in disasters by reviewing existing data sources and compiling new datasets on sex‐differentiated disaster fatalities in the twenty‐first century. The analysis is structured by disaster type, covering geophysical, meteorological, climatological, hydrological,
Olivier Rubin
wiley   +1 more source

CometAnalyser: A user-friendly, open-source deep-learning microscopy tool for quantitative comet assay analysis. [PDF]

open access: yesComput Struct Biotechnol J, 2022
Beleon A   +8 more
europepmc   +1 more source

Corrigendum: Reference cells and ploidy in the comet assay [PDF]

open access: gold, 2017
Gunnar Brunborg   +4 more
openalex   +1 more source

ID3 deficiency alters chromatin accessibility at DSB sites and enhances vulnerability to HDAC inhibition

open access: yesInternational Journal of Cancer, Volume 158, Issue 12, Page 3173-3186, 15 June 2026.
What's new? Errors in DNA double‐strand break (DSB) repair can lead to mutations, chromosomal instability, and ultimately cancer. Inhibitor of DNA‐binding 3 (ID3), a transcriptional repressor, is crucial to promoting DSB repair and helping maintain genome stability. Here, the authors investigated ID3 regulation of DNA repair via chromatin accessibility
Giuditta Della Corte   +10 more
wiley   +1 more source

Redox Regulation and Oxidative Stress in Health and Disease: Mechanisms and Therapeutic Targeting

open access: yesMedComm – Future Medicine, Volume 5, Issue 2, June 2026.
Reactive species serve crucial roles which are tightly regulated in both physiological as well as disease states. At physiological levels, these species are integral to redox signaling, while uncontrolled redox promotes disease pathology. This review examines the dysregulation of these processes.
Mohammad Hossein Azadi   +2 more
wiley   +1 more source

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