Results 101 to 110 of about 3,211,899 (373)

On quasi-complements [PDF]

open access: yesPacific Journal of Mathematics, 1973
has a reflexive, infinite dimensional quotient(cf. [12]).The final result of the paper is that every subspace of a separableconjugate space admits a weak*-closed quasi-complement which isspanned by a boundedly complete w*-basic sequence.The notation and terminology agree with [6].
openaire   +4 more sources

Spatiotemporal and quantitative analyses of phosphoinositides – fluorescent probe—and mass spectrometry‐based approaches

open access: yesFEBS Letters, EarlyView.
Fluorescent probes allow dynamic visualization of phosphoinositides in living cells (left), whereas mass spectrometry provides high‐sensitivity, isomer‐resolved quantitation (right). Their synergistic use captures complementary aspects of lipid signaling. This review illustrates how these approaches reveal the spatiotemporal regulation and quantitative
Hiroaki Kajiho   +3 more
wiley   +1 more source

Therapeutic Lessons to be Learned From the Role of Complement Regulators as Double-Edged Sword in Health and Disease

open access: yesFrontiers in Immunology, 2020
The complement system is an important part of the innate immune system, providing a strong defense against pathogens and removing apoptotic cells and immune complexes.
Esther C. W. de Boer   +4 more
doaj   +1 more source

The anti‐CRISPR protein AcrIE8.1 inhibits the type I‐E CRISPR‐Cas system by directly binding to the Cascade subunit Cas11

open access: yesFEBS Letters, EarlyView.
In this study, we present the structure of AcrIE8.1, a previously uncharacterized anti‐CRISPR protein that inhibits the type I‐E CRISPR‐Cas system. Through a combination of structural and biochemical analyses, we demonstrate that AcrIE8.1 directly binds to the Cas11 subunit of the Cascade complex to inhibit the CRISPR‐Cas system.
Young Woo Kang, Hyun Ho Park
wiley   +1 more source

Transcriptomic atlas reveals organ-specific disease tolerance in sickle cell mice

open access: yesBlood Advances
: Sickle cell disease (SCD) is the most common genetic disease in the world and a societal challenge. SCD is characterized by multiorgan injury related to intravascular hemolysis.
Anne Grunenwald   +9 more
doaj   +1 more source

Hardy inequalities and Assouad dimensions

open access: yes, 2017
We establish both sufficient and necessary conditions for weighted Hardy inequalities in metric spaces in terms of Assouad (co)dimensions. Our sufficient conditions in the case where the complement is thin are new even in Euclidean spaces, while in the ...
Lehrbäck, Juha
core   +1 more source

On Schur's complement

open access: yesJournal of Combinatorial Theory, Series A, 1973
AbstractA new proof is given for Haynsworth's formula for Schur's complement.
openaire   +3 more sources

When is the sum of complemented subspaces complemented? [PDF]

open access: yesStudia Mathematica, 2020
We provide a sufficient condition for the sum of a finite number of complemented subspaces of a Banach space to be complemented. Under this condition a formula for a projection onto the sum is given. We also show that the condition is sharp (in a certain sense).
openaire   +3 more sources

An intracellular transporter mitigates the CO2‐induced decline in iron content in Arabidopsis shoots

open access: yesFEBS Letters, EarlyView.
This study identifies a gene encoding a transmembrane protein, MIC, which contributes to the reduction of shoot Fe content observed in plants under elevated CO2. MIC is a putative Fe transporter localized to the Golgi and endosomal compartments. Its post‐translational regulation in roots may represent a potential target for improving plant nutrition ...
Timothy Mozzanino   +7 more
wiley   +1 more source

Functional Characterization of the Disease-Associated N-Terminal Complement Factor H Mutation W198R

open access: yesFrontiers in Immunology, 2017
Dysregulation of the complement alternative pathway is involved in the pathogenesis of several diseases, including the kidney diseases atypical hemolytic uremic syndrome (aHUS) and C3 glomerulopathy (C3G).
Marcell Cserhalmi   +8 more
doaj   +1 more source

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