Results 161 to 170 of about 40,238 (270)

Advances in Cellular Immune Theranostic Approaches for Glioblastoma: Current Trends and Future Directions

open access: yesCancer Innovation, Volume 4, Issue 4, August 2025.
Glioblastoma (GBM) remains a highly malignant brain tumor with limited treatment efficacy, driving the need for innovative cellular immune theranostic strategies that integrate diagnostic and therapeutic approaches. Recent advances in cellular immunotherapy for GBM include chimeric antigen receptor (CAR)‐T/NK cells, macrophage‐based therapies ...
Ying Gong   +14 more
wiley   +1 more source

Complement C1q-dependent excitatory and inhibitory synapse elimination by astrocytes and microglia in Alzheimer's disease mouse models. [PDF]

open access: yesNat Aging, 2022
Dejanovic B   +21 more
europepmc   +1 more source

The asparagine-linked sugar chains of subcomponent C1q of the first component of human complement.

open access: hybrid, 1978
Tsuguo Mizuochi   +3 more
openalex   +1 more source

The Binding Protein for Globular Heads of Complement C1q, gC1qR [PDF]

open access: hybrid, 1996
Boon Leong Lim   +5 more
openalex   +1 more source

Endothelial Glycocalyx: The Missing Link Between Angiogenic Imbalance in Preeclampsia and Systemic Inflammation in HELLP Syndrome

open access: yesComprehensive Physiology, Volume 15, Issue 4, August 2025.
Endothelial glycocalyx may be the missing link between angiogenic factors, inflammatory regulation, and endothelial maternal dysfunction in early‐onset preeclampsia and HELLP syndrome. ABSTRACT The pathophysiology of preeclampsia and HELLP syndrome relies on systemic vascular endothelial dysfunction, resulting from angiogenic imbalance due to abnormal ...
Anthony Atallah   +4 more
wiley   +1 more source

Microglial IKKβ Alters Central and Peripheral Immune Activity at Distinct Time Points After Spinal Cord Injury

open access: yesGlia, Volume 73, Issue 8, Page 1746-1766, August 2025.
Main Points Microglial NF‐kB signaling increases M1‐like spinal microglia over time after spinal cord injury. Microglial NF‐kB signaling broadly affects gene expression in spinal cord. Microglial NF‐kB signaling modulates splenocytes acutely after spinal cord injury.
Micaela L. O'Reilly   +8 more
wiley   +1 more source

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