Results 201 to 210 of about 268,987 (334)

Complement activation and C3 glomerulopathy

open access: yesJapanese journal of pediatric nephrology, 2022
openaire   +1 more source

Stamping Lithography on Arbitrary Surfaces based on Self‐Assembly of Colloidal Particles

open access: yesAdvanced Science, EarlyView.
A new method called Stamping Lithography for three‐dimensional (3D) circuits manufacturing is proposed, which comprises a resist mask stamping process based on self‐assembly of colloidal particles and a subsequent etching process. It replaces the photoresist with self‐assembled particles and the exposure process with stamping, making it feasible to ...
Guoxu Yu   +8 more
wiley   +1 more source

SIRT5–RAC2 Axis Drives Monocyte‐to‐Macrophage Differentiation to Promote Inflammatory Injury in Premature Ovarian Insufficiency

open access: yesAdvanced Science, EarlyView.
SIRT5 desuccinylates and stabilizes RAC2, activating CSF1R‐dependent signaling to drive monocyte differentiation into M0 macrophages and their polarization toward pro‐inflammatory M1 phenotypes in CTX‐induced premature ovarian insufficiency. Inhibiting the SIRT5‐RAC2 axis attenuates inflammation, reduces granulosa cell apoptosis, and preserves ...
Wenjing TanTai   +15 more
wiley   +1 more source

Complement C3 Activation in the Human Retinal Pigment Epithelium. [PDF]

open access: yesInvest Ophthalmol Vis Sci
Schikora J   +10 more
europepmc   +1 more source

Intracellular Activation of Complement C3 In Paneth Cells Improves Repair of Intestinal Epithelia During Acute Injury [PDF]

open access: hybrid, 2018
Jian Zhang   +8 more
openalex   +1 more source

ROS‐Responsive Wedelolactone Hydrogel Promotes Intervertebral Disc Repair by Disrupting the NF‐κB–LCN2 Inflammatory Feedback Loop

open access: yesAdvanced Science, EarlyView.
A mechanically robust, ROS‐responsive hydrogel is engineered to treat intervertebral disc degeneration by targeting the NF‐κB–LCN2 inflammatory feedback loop. By delivering wedelolactone specifically within oxidative niches, this system disrupts macrophage–disc cell crosstalk.
Zimei Wu   +10 more
wiley   +1 more source

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