Results 141 to 150 of about 273,867 (249)
The impact of nutrition on clinical outcomes in the critically ill child
Luise V. Marino +2 more
openalex +1 more source
Objectives There is growing interest in evaluating new strategies to delay or prevent post‐traumatic osteoarthritis (PTOA) in individuals who have sustained anterior cruciate ligament (ACL) injury. This study sought to determine characteristics of potential treatments that are acceptable to patients with ACL injury.
Kevin Kennedy +9 more
wiley +1 more source
Introduction There are no Food and Drug Administration (FDA) approved therapies for Raynaud's phenomenon (RP) treatment in the United States (U.S.). Clinical trials have been challenged by study design. Important advances in RP patient reported outcome measures and mechanistic quantification allows RP‐related pain characterization.
Tracy M Frech +4 more
wiley +1 more source
Early enteral nutrition in critically-ill patients. [PDF]
Yanamaladoddi V +5 more
europepmc +1 more source
Objective A patient‐centered approach for chronic disease management, including systemic lupus erythematosus (SLE), aligns treatment with patients’ values and preferences, leading to improved outcomes. This paper summarizes how patient experiences, perspectives, and priorities informed the American College of Rheumatology (ACR) 2024 Lupus Nephritis (LN)
Shivani Garg +20 more
wiley +1 more source
Can Nutritional Screening Tools Predict the Prognosis of Critically Ill Patients with Sepsis? [PDF]
Calili DK, Bolukbasi D, Izdes S.
europepmc +1 more source
Corrigendum: Zamberlan P, Feferbaum R, Filho UD, de Carvalho WB, Delgado AF. Bioelectrical impedance phase angle and morbidity and mortality in critically ill children. Nutr Clin Pract. 2019;34(1):163‐171 [PDF]
openalex +1 more source
Beyond Inflammation: Why Understanding the Brain Matters in Inflammatory Arthritis
Persistent pain remains a major challenge in inflammatory arthritis, even when joint inflammation is well controlled. Pain and associated symptoms such as fatigue cannot be explained by peripheral inflammation alone but reflect altered central pain processing. These changes may arise through “top‐down” mechanisms, reflecting pre‐existing dysfunction in
Eoin M. Kelleher +2 more
wiley +1 more source

