Results 171 to 180 of about 29,685 (221)

Diagnostic, Prognostic, and Predictive Molecular Biomarkers in Head and Neck Squamous Cell Carcinoma: A Comprehensive Review. [PDF]

J Clin Med
Michcik A, Wojciechowska B, Tarnawski J, Choma P, Polcyn A, Garbacewicz Ł, Sikora M, Iacoviello P, Wach T, Drogoszewska B.   +9 more
europepmc   +1 more source

Genetic Landscape of Oral Cavity Squamous Cell Carcinoma. [PDF]

OTO Open
Celidonio J, Chinta S, de Armas JS, Roden D.   +3 more
europepmc   +1 more source

Prognostic significance of fibroblast senescence and senescence-associated secretory phenotype factor expression in the tumor microenvironment of pancreatic ductal adenocarcinoma. [PDF]

Oncol Rep
Kitano Y, Miyashita T, Shimasaki T, Makino I, Endo Y, Yamamoto Y, Inaki N, Yagi S.   +7 more
europepmc   +1 more source

Senolytics as Modulators of Critical Signaling Pathways: a Promising Strategy to Combat Brain Aging and Neurodegenerative Disorders. [PDF]

Mol Neurobiol
Singh I, Singh AK.
europepmc   +1 more source

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Expression of the cyclin-dependent kinase inhibitor p16 in Alzheimerʼs disease

NeuroReport, 1996
Recent evidence indicates that an impairment of neuronal adaptive and reparative processes might be a key feature of the pathomechanism of Alzheimer's disease. Aberrations in the reparative neuronal response are likely to result from alterations of intracellular signal transduction cascades.
T, Arendt, L, Rödel, U, Gärtner, M, Holzer   +3 more
openaire   +2 more sources

Receptor-Ck regulates the expression of cyclin-dependent kinase inhibitors (p16; p27)

Molecular and Cellular Biochemistry, 1999
The present study was addressed to understand the interrelationship between Receptor-Ck induction-activation coupling; isoprenoids (derived from mevalonate pathway) and cyclin-dependent kinase inhibitors. The results reported here unambiguously reveal that isoprenoids regulate the expression of genes coding for CDK inhibited p16 and p27.
D, Kaul, M, Kaur
openaire   +2 more sources

p16 Family Inhibitors of Cyclin-Dependent Kinases

1996
The origin and development of human tumors begins at the molecular level and involves a complex multi-step process. A feature common to many tumor cells is their ability to enter and progress through the cell division cycle under conditions where normal cells would either be quiescent or proliferating at a reduced rate.
Yan Li, Christopher W. Jenkins, Michael A. Nichols, Xiaoyu Wu, Kun-Liang Guan, Yue Xiong   +5 more
openaire   +1 more source

Structural integrity of the cyclin‐dependent kinase inhibitor genes, p15, p16 and p18 in myeloid leukaemias

British Journal of Haematology, 1995
Summary The cyclin‐dependent kinase inhibitors known as p15, p16 and p18 have been suggested as candidates for tumour suppressor genes. We examined these genes for their alterations in 46 myeloid leukaemias and 15 myeloid leukaemia cell lines, p16 mRNA expression was studied in 41 myeloid leukaemias. The p15 and p16 genes were either deleted or mutated
Tsuyoshi Nakamaki, Norihiko Kawamata, Juurg Schwaller, Andreas Tobler, Martin Fey, Seppo Pakkala, Young Y. Lee, Byoung K. Kim, Kunihiko Fukuchi, Nobuyoshi Tsuruoka, Jerry Kahan, Carl W. Miller, H. Phillip Koeffler   +12 more
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Cyclin-dependent kinase inhibitors, p16 and p27, demonstrate different expression patterns in thymoma and thymic carcinoma

General Thoracic and Cardiovascular Surgery, 2014
The role of cell cycle inhibitors in tumorigenesis has been proven in various neoplasms; however, their roles in thymic tumors are still unclear. We examined the expression of cell cycle inhibitors such as those of the Cip/Kip family (p21, p27, and p57) and the INK-4/ARF family (p16 and p14) in thymoma and thymic carcinoma.Samples from 41 thymoma and ...
Mutsuko, Omatsu, Toshiaki, Kunimura, Tetsuya, Mikogami, Akira, Shiokawa, Atsuko, Masunaga, Tomoko, Nagai, Akihiko, Kitami, Takashi, Suzuki, Mitsutaka, Kadokura   +8 more
openaire   +2 more sources