The D-type alfalfa cyclin gene cycMs4 complements G1 cyclin-deficient yeast and is induced in the G1 phase of the cell cycle. [PDF]
, 1995 Morten Dahl +7 more
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PD-L1 Expression Fluctuates Concurrently with Cyclin D in Glioblastoma Cells. [PDF]
Cells, 2021 Tufano M +7 more
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Insights from the degradation mechanism of cyclin D into targeted therapy of the cancer cell cycle. [PDF]
Signal Transduct Target Ther, 2021 Caudron-Herger M, Diederichs S.
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D-type cyclins and the DNA damage response
, 2008 The cell cycle is strictly regulated to ensure the precise transmission of genetic information from one cell to its daughter cells. The DNA damage response is one of these regulatory pathways and is activated in response to both intrinsic and extrinsic genotoxic stress.
Suwaki, Natsuko, Suwaki, Natsuko
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DNA tumor virus oncoproteins and retinoblastoma gene mutations share the ability to relieve the cells requirement for cyclin D1 function in G1 [PDF]
, 1994 The retinoblastoma gene product (pRB) participates in the regulation of the cell division cycle through complex formation with numerous cellular regulatory proteins including the potentially oncogenic cyclin D1. Extending the current view of the emerging
Bartek, J. +7 more
core
Selective modulation of the cyclin B/CDK1 and cyclin D/CDK4 complexes during in vitro human megakaryocyte development [PDF]
, 1999 Alessandra Bassini +6 more
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Expression of Concern for Patel et al., "Brk/Protein Tyrosine Kinase 6 Phosphorylates p27KIP1, Regulating the Activity of Cyclin D-Cyclin-Dependent Kinase 4". [PDF]
Mol Cell Biol, 2022 American Society for Microbiology.
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Gene Expression and Cell Cycle Arrest Mediated by Transcription Factor DMP1 Is Antagonized by D-Type Cyclins through a Cyclin-Dependent-Kinase-Independent Mechanism [PDF]
, 1998 Kazushi Inoue, Charles J. Sherr
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On optimality of designs with three distinct eigenvalues [PDF]
arXiv, 2012 Let $\D_{v,b,k}$ denote the family of all connected block designs with $v$ treatments and $b$ blocks of size $k$. Let $d\in\D_{v,b,k}$. The replication of a treatment is the number of times it appears in the blocks of $d$. The matrix $C(d)=R(d)-\frac{1}{k}N(d)N(d)^\top$ is called the information matrix of $d$ where $N(d)$ is the incidence matrix of $d$
arxiv
Cyclin A/CDK2 Regulates V(D)J Recombination by Coordinating RAG-2 Accumulation and DNA Repair [PDF]
, 1999 Jinhak Lee, Stephen Desiderio
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